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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

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Showing 25 citing articles:

Cannabis and adolescent brain development
Dan I. Lubman, Ali Cheetham, Murat Yücel
Pharmacology & Therapeutics (2014) Vol. 148, pp. 1-16
Closed Access | Times Cited: 314
The Impact of Exposure to Cannabinoids in Adolescence: Insights From Animal Models
Tiziana Rubino, Daniela Parolaro
Biological Psychiatry (2015) Vol. 79, Iss. 7, pp. 578-585
Closed Access | Times Cited: 164
Brain-Derived Neurotrophic Factor (BDNF) Role in Cannabinoid-Mediated Neurogenesis
Filipa F. Ferreira, Filipa F. Ribeiro, Rui S. Rodrigues, et al.
Frontiers in Cellular Neuroscience (2018) Vol. 12
Open Access | Times Cited: 77
Schizophrenia and Depression Co-Morbidity: What We have Learned from Animal Models
James Samsom, Albert H.C. Wong
Frontiers in Psychiatry (2015) Vol. 6
Open Access | Times Cited: 73
The dual hit hypothesis of schizophrenia: Evidence from animal models
Cyprien G.J. Guerrin, Janine Doorduin, Iris E. Sommer, et al.
Neuroscience & Biobehavioral Reviews (2021) Vol. 131, pp. 1150-1168
Open Access | Times Cited: 54
Sex differences in animal models of schizophrenia shed light on the underlying pathophysiology
Rachel Hill
Neuroscience & Biobehavioral Reviews (2015) Vol. 67, pp. 41-56
Closed Access | Times Cited: 54
The tyrosine kinase inhibitor, sunitinib malate, induces cognitive impairment in vivo via dysregulating VEGFR signaling, apoptotic and autophagic machineries
Amal Kamal Abdel‐Aziz, Eman M. Mantawy, Riham S. Said, et al.
Experimental Neurology (2016) Vol. 283, pp. 129-141
Closed Access | Times Cited: 50
Sensorimotor gating deficits in “two-hit” models of schizophrenia risk factors
Asma Khan, Susan B. Powell
Schizophrenia Research (2017) Vol. 198, pp. 68-83
Open Access | Times Cited: 40
Analyzing the influence of BDNF heterozygosity on spatial memory response to 17β-estradiol
Y. W. C. Wu, Xin Du, Maarten van den Buuse, et al.
Translational Psychiatry (2015) Vol. 5, Iss. 1, pp. e498-e498
Open Access | Times Cited: 35
Brain-derived neurotrophic factor heterozygous mutant rats show selective cognitive changes and vulnerability to chronic corticosterone treatment
Anand Gururajan, Rachel Hill, Maarten van den Buuse
Neuroscience (2014) Vol. 284, pp. 297-310
Closed Access | Times Cited: 32
Schizophrenia: a consequence of gene-environment interactions?
Tim Karl, Jonathon C. Arnold
Frontiers in Behavioral Neuroscience (2014) Vol. 8
Open Access | Times Cited: 28
BDNF haploinsufficiency induces behavioral endophenotypes of schizophrenia in male mice that are rescued by enriched environment
Mahmoud Harb, Justina Jagusch, Archana Durairaja, et al.
Translational Psychiatry (2021) Vol. 11, Iss. 1
Open Access | Times Cited: 16
Corticosterone treatment during adolescence induces down-regulation of reelin and NMDA receptor subunit GLUN2C expression only in male mice: implications for schizophrenia
Laetitia Buret, Maarten van den Buuse
The International Journal of Neuropsychopharmacology (2014) Vol. 17, Iss. 08, pp. 1221-1232
Open Access | Times Cited: 21
Developmentally vitamin D-deficient rats show enhanced prepulse inhibition after acute Δ9-tetrahydrocannabinol
Thomas H.J. Burne, Suzanne Alexander, Karly M. Turner, et al.
Behavioural Pharmacology (2014) Vol. 25, Iss. 3, pp. 236-244
Closed Access | Times Cited: 14
Neuron-specific deletion of VEGF or its receptor Flk-1 impairs recognition memory
Satoshi Deyama, Xiaoyuan Li, Ronald S. Duman
European Neuropsychopharmacology (2020) Vol. 31, pp. 145-151
Closed Access | Times Cited: 13
Interaction of reelin and stress on immobility in the forced swim test but not dopamine-mediated locomotor hyperactivity or prepulse inhibition disruption: Relevance to psychotic and mood disorders
Michael Notaras, Billie Vivian, Carey Wilson, et al.
Schizophrenia Research (2017) Vol. 215, pp. 485-492
Closed Access | Times Cited: 14
Does genetic BDNF deficiency in rats interact with neurotransmitter control of prepulse inhibition? Implications for schizophrenia
Maarten van den Buuse, Davina Biel, Kathrin Radscheit
Progress in Neuro-Psychopharmacology and Biological Psychiatry (2017) Vol. 75, pp. 192-198
Closed Access | Times Cited: 13
Marijuana use among adolescents is associated with deleterious alterations in mature BDNF
María José Míguez, Wenyaw Chan, Luis Espinoza, et al.
AIMS Public Health (2019) Vol. 6, Iss. 1, pp. 4-14
Open Access | Times Cited: 12
Effect of adolescent androgen manipulation on psychosis-like behaviour in adulthood in BDNF heterozygous and control mice
Xin Du, Cushla R. McCarthny, Michael Notaras, et al.
Hormones and Behavior (2019) Vol. 112, pp. 32-41
Closed Access | Times Cited: 6
Molecular Basis of Cannabis-Induced Schizophrenia-Relevant Behaviours: Insights from Animal Models
Rose Chesworth, Tim Karl
Current Behavioral Neuroscience Reports (2017) Vol. 4, Iss. 3, pp. 254-279
Closed Access | Times Cited: 4
Cannabinoid Modulation of Object Recognition and Location Memory—A Preclinical Assessment
Rose Chesworth, Georgia Watt, Tim Karl
Handbook of behavioral neuroscience (2018), pp. 461-488
Closed Access | Times Cited: 4
‘Two Hit’ Neurodevelopmental Mechanisms in Schizophrenia: Focus on Animal Models and the Role of BDNF
Maren Klug, Rachel Hill, Maarten van den Buuse
Springer eBooks (2015), pp. 335-351
Closed Access | Times Cited: 2
Susceptibility to Psychiatric Diseases After Cannabis Abuse in Adolescence: Animal Models
Tiziana Rubino, Daniela Parolaro
Springer eBooks (2017), pp. 237-255
Closed Access | Times Cited: 1
The dual hit hypothesis of schizophrenia
Cyprien G.J. Guerrin
(2023)
Open Access
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