OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Mitochondrial Injury and Targeted Intervention in Septic Cardiomyopathy
Ying Tan, Sainan Chen, Jiankai Zhong, et al.
Current Pharmaceutical Design (2019) Vol. 25, Iss. 18, pp. 2060-2070
Closed Access | Times Cited: 43

Showing 1-25 of 43 citing articles:

TMBIM6 prevents VDAC1 multimerization and improves mitochondrial quality control to reduce sepsis-related myocardial injury
Hao Zhou, Zhe Dai, Jialei Li, et al.
Metabolism (2023) Vol. 140, pp. 155383-155383
Closed Access | Times Cited: 55

Current Status of Septic Cardiomyopathy: Basic Science and Clinical Progress
Huan Lin, Wenting Wang, Madeline Lee, et al.
Frontiers in Pharmacology (2020) Vol. 11
Open Access | Times Cited: 91

Mitochondrial Ca2+ regulation in the etiology of heart failure: physiological and pathophysiological implications
Haixia Xu, Sumei Cui, Yingmei Zhang, et al.
Acta Pharmacologica Sinica (2020) Vol. 41, Iss. 10, pp. 1301-1309
Open Access | Times Cited: 81

lncRNA RMRP Prevents Mitochondrial Dysfunction and Cardiomyocyte Apoptosis via the miR-1-5p/hsp70 Axis in LPS-Induced Sepsis Mice
Ying Han, Yixin Cai, Xiaoquan Lai, et al.
Inflammation (2020) Vol. 43, Iss. 2, pp. 605-618
Closed Access | Times Cited: 71

S100a8/a9 contributes to sepsis-induced cardiomyopathy by activating ERK1/2-Drp1-mediated mitochondrial fission and respiratory dysfunction
Feng Wu, Yanting Zhang, Fei Teng, et al.
International Immunopharmacology (2023) Vol. 115, pp. 109716-109716
Open Access | Times Cited: 37

Cardiac-specific overexpression of catalase attenuates lipopolysaccharide-induced cardiac anomalies through reconciliation of autophagy and ferroptosis
Peng Hu, Ji Zhang, Zhonglin Zhang, et al.
Life Sciences (2023) Vol. 328, pp. 121821-121821
Closed Access | Times Cited: 35

Curcumin simultaneously improves mitochondrial dynamics and myocardial cell bioenergy after sepsis via the SIRT1-DRP1/PGC-1α pathway
Dongyao Hou, Haitang Liao, Shuai Hao, et al.
Heliyon (2024) Vol. 10, Iss. 7, pp. e28501-e28501
Open Access | Times Cited: 9

Targeting ferroptosis in the maintenance of mitochondrial homeostasis in the realm of septic cardiomyopathy
Hua Ye, Huantao Hu, Xiaoliang Zhou, et al.
Current Opinion in Pharmacology (2024) Vol. 74, pp. 102430-102430
Closed Access | Times Cited: 8

Cardamonin protects against lipopolysaccharide-induced myocardial contractile dysfunction in mice through Nrf2-regulated mechanism
Ying Tan, Honghong Wan, Mingming Sun, et al.
Acta Pharmacologica Sinica (2020) Vol. 42, Iss. 3, pp. 404-413
Open Access | Times Cited: 55

Dual specificity phosphatase 1 attenuates inflammation-induced cardiomyopathy by improving mitophagy and mitochondrial metabolism
Ying Tan, Yue Zhang, Jing He, et al.
Molecular Metabolism (2022) Vol. 64, pp. 101567-101567
Open Access | Times Cited: 30

ALDH2 mitigates LPS-induced cardiac dysfunction, inflammation, and apoptosis through the cGAS/STING pathway
H. Liu, Qin Hu, Ke Ren, et al.
Molecular Medicine (2023) Vol. 29, Iss. 1
Open Access | Times Cited: 20

Clinical implications of septic cardiomyopathy: A narrative review
Hiroaki Hiraiwa, Daisuke Kasugai, Takahiro Okumura, et al.
Medicine (2024) Vol. 103, Iss. 17, pp. e37940-e37940
Open Access | Times Cited: 7

Long Non-Coding RNAs as Biomarkers and Therapeutic Targets in Sepsis
Chuqiao Wang, Guorui Liang, Jieni Shen, et al.
Frontiers in Immunology (2021) Vol. 12
Open Access | Times Cited: 35

DUSP1 interacts with and dephosphorylates VCP to improve mitochondrial quality control against endotoxemia-induced myocardial dysfunction
Hang Zhu, Jin Wang, Ting Xin, et al.
Cellular and Molecular Life Sciences (2023) Vol. 80, Iss. 8
Closed Access | Times Cited: 16

Inhibition of miR‑101‑3p protects against sepsis‑induced myocardial injury by inhibiting MAPK and NF‑κB pathway activation via the upregulation of DUSP1
Xin Ye, Li Tang, Jing Chen, et al.
International Journal of Molecular Medicine (2021) Vol. 47, Iss. 3
Open Access | Times Cited: 32

CircTLK1 modulates sepsis‐induced cardiomyocyte apoptosis via enhancing PARP1/HMGB1 axis–mediated mitochondrial DNA damage by sponging miR‐17‐5p
Yu Qiu, Ying Yu, Xiaomei Qin, et al.
Journal of Cellular and Molecular Medicine (2021) Vol. 25, Iss. 17, pp. 8244-8260
Open Access | Times Cited: 27

CD74 knockout protects against LPS‐induced myocardial contractile dysfunction through AMPK‐Skp2‐SUV39H1‐mediated demethylation of BCLB
Yuanfei Luo, Congcong Fan, Mingjie Yang, et al.
British Journal of Pharmacology (2019) Vol. 177, Iss. 8, pp. 1881-1897
Open Access | Times Cited: 34

GAS6 attenuates sepsis-induced cardiac dysfunction through NLRP3 inflammasome-dependent mechanism
Ting Ji, Qiong Liu, Liming Yu, et al.
Free Radical Biology and Medicine (2023) Vol. 210, pp. 195-211
Closed Access | Times Cited: 10

Ablation of Mitophagy Receptor FUNDC1 Accentuates Septic Cardiomyopathy through ACSL4-Dependent Regulation of Ferroptosis and Mitochondrial Integrity
Fengjuan Li, Huantao Hu, Liangyan Wu, et al.
Free Radical Biology and Medicine (2024) Vol. 225, pp. 75-86
Closed Access | Times Cited: 3

The role of the cholinergic anti-inflammatory pathway in septic cardiomyopathy
Wenting Wang, Hui Xu, Huan Lin, et al.
International Immunopharmacology (2020) Vol. 90, pp. 107160-107160
Closed Access | Times Cited: 25

Rosmarinic acid protects against lipopolysaccharide‐induced cardiac dysfunction via activating Sirt1/PGC‐1α pathway to alleviate mitochondrial impairment
Ke Peng, Fengyuan Yang, Chenming Qiu, et al.
Clinical and Experimental Pharmacology and Physiology (2022) Vol. 50, Iss. 3, pp. 218-227
Closed Access | Times Cited: 14

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