
OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!
If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.
Requested Article:
Dysgenesis and Dysfunction of the Pancreas and Pituitary Due toFOXA2Gene Defects
Sare Betül Kaygusuz, Esra Arslan Ateş, Maria Lillina Vignola, et al.
The Journal of Clinical Endocrinology & Metabolism (2021) Vol. 106, Iss. 10, pp. e4142-e4154
Open Access | Times Cited: 9
Sare Betül Kaygusuz, Esra Arslan Ateş, Maria Lillina Vignola, et al.
The Journal of Clinical Endocrinology & Metabolism (2021) Vol. 106, Iss. 10, pp. e4142-e4154
Open Access | Times Cited: 9
Showing 9 citing articles:
Syndromic forms of congenital hyperinsulinism
Martin Zenker, Klaus Mohnike, Katja Palm
Frontiers in Endocrinology (2023) Vol. 14
Open Access | Times Cited: 13
Martin Zenker, Klaus Mohnike, Katja Palm
Frontiers in Endocrinology (2023) Vol. 14
Open Access | Times Cited: 13
Chromosome 20p11.2 deletions cause congenital hyperinsulinism via the loss of FOXA2 or its regulatory elements
Thomas W. Laver, Matthew N. Wakeling, Richard Caswell, et al.
European Journal of Human Genetics (2024) Vol. 32, Iss. 7, pp. 813-818
Open Access | Times Cited: 4
Thomas W. Laver, Matthew N. Wakeling, Richard Caswell, et al.
European Journal of Human Genetics (2024) Vol. 32, Iss. 7, pp. 813-818
Open Access | Times Cited: 4
Large copy number variants are an important cause of congenital hyperinsulinism that should be screened for during routine testing
Sarah E. Flanagan, Isabella-Anna Lazaridi, Jonna M. E. Männistö, et al.
Frontiers in Endocrinology (2025) Vol. 16
Open Access
Sarah E. Flanagan, Isabella-Anna Lazaridi, Jonna M. E. Männistö, et al.
Frontiers in Endocrinology (2025) Vol. 16
Open Access
Re-analysis of gene mutations found in pituitary stalk interruption syndrome and a new hypothesis on the etiology
Shengjie Wang, Qiaozhen Qin, Deyue Jiang, et al.
Frontiers in Endocrinology (2024) Vol. 15
Open Access | Times Cited: 2
Shengjie Wang, Qiaozhen Qin, Deyue Jiang, et al.
Frontiers in Endocrinology (2024) Vol. 15
Open Access | Times Cited: 2
Novel genes and variants associated with congenital pituitary hormone deficiency in the era of next-generation sequencing
Hironori Bando, Shin Urai, Keitaro Kanie, et al.
Frontiers in Endocrinology (2022) Vol. 13
Open Access | Times Cited: 10
Hironori Bando, Shin Urai, Keitaro Kanie, et al.
Frontiers in Endocrinology (2022) Vol. 13
Open Access | Times Cited: 10
Congenital hyperinsulinism and panhypopituitarism: a rare combination
Foram Patel, Ginger Darling, Ahmed Torky
Endocrinology Diabetes and Metabolism Case Reports (2023) Vol. 2023, Iss. 2
Open Access | Times Cited: 2
Foram Patel, Ginger Darling, Ahmed Torky
Endocrinology Diabetes and Metabolism Case Reports (2023) Vol. 2023, Iss. 2
Open Access | Times Cited: 2
PIAS1 upregulation confers protection against Cerulein-induced acute pancreatitis via FTO downregulation by enhancing sumoylation of Foxa2
Jiandang Zhou, Qiao Wen Tan, Jinxue Tong, et al.
Genomics (2023) Vol. 115, Iss. 5, pp. 110693-110693
Open Access | Times Cited: 2
Jiandang Zhou, Qiao Wen Tan, Jinxue Tong, et al.
Genomics (2023) Vol. 115, Iss. 5, pp. 110693-110693
Open Access | Times Cited: 2
Congenital hyperinsulinism in a newborn presenting with poor feeding
Kiran Mazloom, Pedro A. Sanchez‐Lara, Seth Langston, et al.
SAGE Open Medical Case Reports (2022) Vol. 10, pp. 2050313X2210831-2050313X2210831
Open Access | Times Cited: 1
Kiran Mazloom, Pedro A. Sanchez‐Lara, Seth Langston, et al.
SAGE Open Medical Case Reports (2022) Vol. 10, pp. 2050313X2210831-2050313X2210831
Open Access | Times Cited: 1
Chromosome 20p11.2 deletions cause congenital hyperinsulinism via the likely disruption ofFOXA2
Thomas W. Laver, Matthew N. Wakeling, Richard Caswell, et al.
medRxiv (Cold Spring Harbor Laboratory) (2023)
Open Access
Thomas W. Laver, Matthew N. Wakeling, Richard Caswell, et al.
medRxiv (Cold Spring Harbor Laboratory) (2023)
Open Access