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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Dysfunction in nonsense-mediated decay, protein homeostasis, mitochondrial function, and brain connectivity in ALS-FUS mice with cognitive deficits
Wan Yun Ho, Ira Agrawal, Sheue-Houy Tyan, et al.
Acta Neuropathologica Communications (2021) Vol. 9, Iss. 1
Open Access | Times Cited: 24

Showing 24 citing articles:

Molecular hallmarks of ageing in amyotrophic lateral sclerosis
Cyril J. Jagaraj, Sina Shadfar, Sara Assar Kashani, et al.
Cellular and Molecular Life Sciences (2024) Vol. 81, Iss. 1
Open Access | Times Cited: 11
Detection of Brain Somatic Mutations in Cerebrospinal Fluid from Refractory Epilepsy Patients
Seyeon Kim, Sara Baldassari, Nam Suk Sim, et al.
Annals of Neurology (2021) Vol. 89, Iss. 6, pp. 1248-1252
Closed Access | Times Cited: 46
Synaptic dysfunction in ALS and FTD: anatomical and molecular changes provide insights into mechanisms of disease
Pauline A. Gelon, Paul A. Dutchak, Chantelle F. Sephton
Frontiers in Molecular Neuroscience (2022) Vol. 15
Open Access | Times Cited: 35
Synaptic Dysfunction and Plasticity in Amyotrophic Lateral Sclerosis
Rosario Gulino
International Journal of Molecular Sciences (2023) Vol. 24, Iss. 5, pp. 4613-4613
Open Access | Times Cited: 21
Longitudinal multi-omics in alpha-synuclein Drosophila model discriminates disease- from age-associated pathologies in Parkinson’s disease
Justin Moore, Timothy Wu, Justin Dhindsa, et al.
npj Parkinson s Disease (2025) Vol. 11, Iss. 1
Open Access
Current Concepts on Genetic Aspects of Mitochondrial Dysfunction in Amyotrophic Lateral Sclerosis
Milena Janković, Ivana Novaković, Phepy G. A. Dawod, et al.
International Journal of Molecular Sciences (2021) Vol. 22, Iss. 18, pp. 9832-9832
Open Access | Times Cited: 28
The Spectrum of Cognitive Dysfunction in Amyotrophic Lateral Sclerosis: An Update
K. A. Jellinger
International Journal of Molecular Sciences (2023) Vol. 24, Iss. 19, pp. 14647-14647
Open Access | Times Cited: 12
Reactive astrocytes in ALS display diminished intron retention
Oliver J. Ziff, Doaa M. Taha, Hamish Crerar, et al.
Nucleic Acids Research (2021) Vol. 49, Iss. 6, pp. 3168-3184
Open Access | Times Cited: 26
Neuronal dysfunction caused by FUSR521G promotes ALS-associated phenotypes that are attenuated by NF-κB inhibition
Mari Carmen Pelaez, Antoine Desmeules, Pauline A. Gelon, et al.
Acta Neuropathologica Communications (2023) Vol. 11, Iss. 1
Open Access | Times Cited: 8
Disruption of orbitofrontal-hypothalamic projections in a murine ALS model and in human patients
David Bayer, Stefano Antonucci, Hans‐Peter Müller, et al.
Translational Neurodegeneration (2021) Vol. 10, Iss. 1
Open Access | Times Cited: 19
The Interplay of RNA Binding Proteins, Oxidative Stress and Mitochondrial Dysfunction in ALS
Jasmine Harley, Benjamin Clarke, Rickie Patani
Antioxidants (2021) Vol. 10, Iss. 4, pp. 552-552
Open Access | Times Cited: 18
Studies of Genetic and Proteomic Risk Factors of Amyotrophic Lateral Sclerosis Inspire Biomarker Development and Gene Therapy
Eva Bagyinszky, John Hulme, Seong Soo A. An
Cells (2023) Vol. 12, Iss. 15, pp. 1948-1948
Open Access | Times Cited: 7
Reversal of cognitive deficits in FUSR521G amyotrophic lateral sclerosis mice by arimoclomol and a class I histone deacetylase inhibitor independent of heat shock protein induction
Mari Carmen Pelaez, Frédéric Di Fiore, Nancy Larochelle, et al.
Neurotherapeutics (2024) Vol. 21, Iss. 5, pp. e00388-e00388
Closed Access | Times Cited: 2
Gelatin methacrylate hydrogels culture model for glioblastoma cells enriches for mesenchymal-like state and models interactions with immune cells
Nameeta Shah, Pavan M. Hallur, Raksha A. Ganesh, et al.
Scientific Reports (2021) Vol. 11, Iss. 1
Open Access | Times Cited: 16
From use of omics to systems biology: Identifying therapeutic targets for amyotrophic lateral sclerosis
Lydia M. Castelli, Rosario Vasta, Scott P. Allen, et al.
International review of neurobiology (2024), pp. 209-268
Closed Access | Times Cited: 1
A postzygotic de novo NCDN mutation identified in a sporadic FTLD patient results in neurochondrin haploinsufficiency and altered FUS granule dynamics
Gaël Nicolas, Myriam Sévigny, François Lecoquierre, et al.
Acta Neuropathologica Communications (2022) Vol. 10, Iss. 1
Open Access | Times Cited: 7
Elucidation of the Landscape of Alternatively Spliced Genes and Features in the Dorsal Striatum of Aggressive/Aggression-Deprived Mice in the Model of Chronic Social Conflicts
Vladimir N. Babenko, Olga Redina, Dmitry A. Smagin, et al.
Genes (2023) Vol. 14, Iss. 3, pp. 599-599
Open Access | Times Cited: 3
FUS-dependent microRNA deregulations identify TRIB2 as a druggable target for ALS motor neurons
Wan Yun Ho, Li-Ling Chak, Jin‐Hui Hor, et al.
iScience (2023) Vol. 26, Iss. 11, pp. 108152-108152
Open Access | Times Cited: 3
Proximity-based labeling reveals DNA damage–induced phosphorylation of fused in sarcoma (FUS) causes distinct changes in the FUS protein interactome
Michelle A. Johnson, Thomas A. Nuckols, Paola Merino, et al.
Journal of Biological Chemistry (2022) Vol. 298, Iss. 8, pp. 102135-102135
Open Access | Times Cited: 3
Animal Models of FUS-Proteinopathy: A Systematic Review
Alexander P. Rezvykh, Daniil Shteinberg, Evgeny V. Bronovitsky, et al.
Biochemistry (Moscow) (2024) Vol. 89, Iss. S1, pp. S34-S56
Closed Access
Liquid–liquid phase separation in presynaptic nerve terminals
Ji-Young Choi, Nisha Mohd Rafiq, Daehun Park
Trends in Biochemical Sciences (2024) Vol. 49, Iss. 10, pp. 888-900
Closed Access
Mild cognitive impairment in amyotrophic lateral sclerosis: current view
K. A. Jellinger
Journal of Neural Transmission (2024)
Closed Access
Stress Granules, a Novel Therapeutic Target of FUS-Related Amyotrophic Lateral Sclerosis
钰林 周
Hans Journal of Biomedicine (2022) Vol. 12, Iss. 02, pp. 55-65
Closed Access | Times Cited: 1
Proximity-based labeling reveals DNA damage-induced N-terminal phosphorylation of fused in sarcoma (FUS) leads to distinct changes in the FUS protein interactome
Michelle A. Johnson, Thomas A. Nuckols, Paola Merino, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2021)
Open Access
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