OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Inflammasome inhibition protects dopaminergic neurons from α-synuclein pathology in a model of progressive Parkinson’s disease
Alexander Grotemeyer, Judith F. Fischer, James B. Koprich, et al.
Journal of Neuroinflammation (2023) Vol. 20, Iss. 1
Open Access | Times Cited: 18

Showing 18 citing articles:

Metabolic reprogramming and polarization of microglia in Parkinson’s disease: Role of inflammasome and iron
Haiyang Yu, Qing Chang, Tong Sun, et al.
Ageing Research Reviews (2023) Vol. 90, pp. 102032-102032
Closed Access | Times Cited: 51

Mitochondria Dysfunction and Neuroinflammation in Neurodegeneration: Who Comes First?
Caterina Peggion, Tito Calì, Marisa Brini
Antioxidants (2024) Vol. 13, Iss. 2, pp. 240-240
Open Access | Times Cited: 22

Innate immune activation in neurodegenerative diseases
Sergio Castro‐Gomez, Michael T. Heneka
Immunity (2024) Vol. 57, Iss. 4, pp. 790-814
Closed Access | Times Cited: 14

Targeting NLRP3 inflammasome for neurodegenerative disorders
Jing Yao, Zhe Wang, Weihong Song, et al.
Molecular Psychiatry (2023) Vol. 28, Iss. 11, pp. 4512-4527
Closed Access | Times Cited: 29

Potential convergence of olfactory dysfunction in Parkinson’s disease and COVID-19: The role of neuroinflammation
Hui Li, Junliang Qian, Youcui Wang, et al.
Ageing Research Reviews (2024) Vol. 97, pp. 102288-102288
Closed Access | Times Cited: 6

Role of Peripheral NLRP3 Inflammasome in Cognitive Impairments: Insights of Non-central Factors
Min Qiao, Junjun Ni, Hong Qing, et al.
Molecular Neurobiology (2025)
Closed Access

Tissue macrophages: origin, heterogenity, biological functions, diseases and therapeutic targets
Fan Guan, Ruixuan Wang, Zhenjie Yi, et al.
Signal Transduction and Targeted Therapy (2025) Vol. 10, Iss. 1
Open Access

Metal–Organic Framework Based Nanozyme System for NLRP3 Inflammasome‐Mediated Neuroinflammatory Regulation in Parkinson's Disease
Qing Li, Xin Ding, Zhaohui Chang, et al.
Advanced Healthcare Materials (2023) Vol. 13, Iss. 10
Open Access | Times Cited: 13

Standardized wireless deep brain stimulation system for mice
Alexander Grotemeyer, Tobias Petschner, Robert L. Peach, et al.
npj Parkinson s Disease (2024) Vol. 10, Iss. 1
Open Access | Times Cited: 1

Inflammasomes in neurodegenerative diseases
Qianchen Wang, Songwei Yang, Xuan Zhang, et al.
Translational Neurodegeneration (2024) Vol. 13, Iss. 1
Open Access | Times Cited: 1

Design, synthesis and evaluation of monoketene compounds as novel potential Parkinson's disease agents by suppressing ER stress via AKT
Jun Chen, Zhiwei Zheng, Mingqi Li, et al.
Bioorganic Chemistry (2023) Vol. 136, pp. 106543-106543
Closed Access | Times Cited: 4

Pyroptosis: the dawn of a new era in endometrial cancer treatment
Peng Tian, Chi Zhang, Wenjun Chen, et al.
Frontiers in Oncology (2023) Vol. 13
Open Access | Times Cited: 4

Use of antiviral drugs and incidence of Parkinson’s disease in Taiwan
Hanwei Zhang, Jun Xie, Hsiao-Ching Peng, et al.
PLoS ONE (2024) Vol. 19, Iss. 5, pp. e0302383-e0302383
Open Access

Pyroptosis mediated by Parkin-NLRP3 negative feedback loop contributed to Parkinson’s disease induced by rotenone
Dongyan Zheng, Yixi Lai, Kai-Lun Huang, et al.
International Immunopharmacology (2024) Vol. 143, pp. 113608-113608
Closed Access

Inosine exerts dopaminergic neuroprotective effects via mitigation of NLRP3 inflammasome activation
Khanal Shristi, Eun‐Joo Shin, Chang Jae Yoo, et al.
Neuropharmacology (2024), pp. 110278-110278
Closed Access

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