OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

N-glycosylation acts as a switch for FGFR1 trafficking between the plasma membrane and nuclear envelope
Paulina Gregorczyk, Natalia Porębska, Dominika Żukowska, et al.
Cell Communication and Signaling (2023) Vol. 21, Iss. 1
Open Access | Times Cited: 5

Showing 5 citing articles:

Glycosylation of FGF/FGFR: An underrated sweet code regulating cellular signaling programs
Aleksandra Gędaj, Paulina Gregorczyk, Dominika Żukowska, et al.
Cytokine & Growth Factor Reviews (2024) Vol. 77, pp. 39-55
Open Access | Times Cited: 5

Exploring Retrograde Trafficking: Mechanisms and Consequences in Cancer and Disease
Rachel Bingham, Helen O. McCarthy, Niamh E. Buckley
Traffic (2024) Vol. 25, Iss. 2
Open Access | Times Cited: 3

The Complexity and Significance of Fibroblast Growth Factor (FGF) Signaling for FGF-Targeted Cancer Therapies
A. Nguyen, Caroline O. Facey, Bruce M. Boman
Cancers (2024) Vol. 17, Iss. 1, pp. 82-82
Open Access | Times Cited: 1

The intracellular interplay between galectin-1 and FGF12 in the assembly of ribosome biogenesis complex
Aleksandra Gędaj, Aleksandra Chorążewska, Krzysztof Ciura, et al.
Cell Communication and Signaling (2024) Vol. 22, Iss. 1
Open Access

GPNMB is a novel binding partner of FGFR1 that affects tumorigenic potential through AKT phosphorylation in TNBC
Manar A. Elhinnawi, Yukari Okita, Kunihiko Shigematsu, et al.
Cancer Science (2024) Vol. 116, Iss. 2, pp. 432-443
Closed Access

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