Open Access Helper

OpenAlex Citation Counts

OpenAlex Citations Logo

OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Effect of TLR4/MyD88/NF‐kB axis in paraventricular nucleus on ventricular arrhythmias induced by sympathetic hyperexcitation in post‐myocardial infarction rats
Kang Wang, Shuling You, Hesheng Hu, et al.
Journal of Cellular and Molecular Medicine (2022) Vol. 26, Iss. 10, pp. 2959-2971
Open Access | Times Cited: 21

Showing 21 citing articles:

Luteolin Attenuates Hypertension via Inhibiting NF-κB-Mediated Inflammation and PI3K/Akt Signaling Pathway in the Hypothalamic Paraventricular Nucleus
Hong‐Li Gao, Xiao‐Jing Yu, Yu‐Qi Feng, et al.
Nutrients (2023) Vol. 15, Iss. 3, pp. 502-502
Open Access | Times Cited: 30
Amelioration of endothelial integrity by 3,5,4′-trihydroxy-trans-stilbene against high-fat-diet-induced obesity and -associated vasculopathy and myocardial infarction in rats, targeting TLR4/MyD88/NF-κB/iNOS signaling cascade
Jitender Singh, Priyanka Bisht, Srishti Srivastav, et al.
Biochemical and Biophysical Research Communications (2024) Vol. 705, pp. 149756-149756
Closed Access | Times Cited: 6
Central control of cardiac activity as assessed by intra-cerebral recordings and stimulations
Laure Mazzola, François Mauguı̀ere, Florian Chouchou
Neurophysiologie Clinique (2023) Vol. 53, Iss. 2, pp. 102849-102849
Open Access | Times Cited: 12
Maresin1 ameliorates ventricular remodelling and arrhythmia in mice models of myocardial infarction via NRF2/HO-1 and TLR4/NF-kB signalling
Fengyuan Wang, Gong Yang, Tao Chen, et al.
International Immunopharmacology (2022) Vol. 113, pp. 109369-109369
Closed Access | Times Cited: 13
Mesenchymal Stem Cell-Derived Exosomes Ameliorate Doxorubicin-Induced Cardiotoxicity
Sawdah Afiyah Ali, Dinender K. Singla
Pharmaceuticals (2024) Vol. 17, Iss. 1, pp. 93-93
Open Access | Times Cited: 2
Suppression of NLRP3 inflammasome orchestrates the protective efficacy of tiron against isoprenaline-induced myocardial injury
Doaa Abdelrahaman, Ola A. Habotta, Ehab S. Taher, et al.
Frontiers in Pharmacology (2024) Vol. 15
Open Access | Times Cited: 2
Inhibition of Mogroside IIIE on isoproterenol-induced myocardial fibrosis through the TLR4/MyD88/NF-κB signaling pathway.
Yanan Shi, Bohan Li, Sun Shuaifeng, et al.
DOAJ (DOAJ: Directory of Open Access Journals) (2023) Vol. 26, Iss. 1, pp. 114-120
Closed Access | Times Cited: 4
The active phthalate metabolite, DHEP, induces proliferation and metastasis of prostate cancer cells via upregulation of β-catenin and downregulation of KLF7
Hecheng Li, Jianping Li, Yubo Ma, et al.
Bioorganic Chemistry (2023) Vol. 141, pp. 106864-106864
Closed Access | Times Cited: 4
Autonomic Neuronal Modulations in Cardiac Arrhythmias: Current Concepts and Emerging Therapies
Ravina Rai, Virendra Singh, Zaved Ahmad, et al.
Physiology & Behavior (2024) Vol. 279, pp. 114527-114527
Closed Access | Times Cited: 1
Endoplasmic reticulum stress-activated nuclear factor-kappa B signaling pathway induces the upregulation of cardiomyocyte dopamine D1 receptor in heart failure
Shun Nakamura, Genri Numata, Toshihiro Yamaguchi, et al.
Biochemical and Biophysical Research Communications (2022) Vol. 637, pp. 247-253
Closed Access | Times Cited: 7
Effect of Perilla frutescens polyphenols against tight junction alterations and intestinal inflammation: a Caco-2/macrophage co-culture model-based study integrated with network pharmacology
Anqi Hu, Yuhui Ye, Binsong Han, et al.
Food Bioscience (2024) Vol. 62, pp. 105459-105459
Closed Access | Times Cited: 1
lncRNA LOC100911717-targeting GAP43-mediated sympathetic remodeling after myocardial infarction in rats
Pingjiang Li, Kang Wang, Jie Yin, et al.
Frontiers in Cardiovascular Medicine (2023) Vol. 9
Open Access | Times Cited: 3
Gab1 Overexpression Attenuates Susceptibility to Ventricular Arrhythmias in Pressure Overloaded Heart Failure Mouse Hearts
Mingxin Liu, Jianhua Zhao, Yonghua Lü, et al.
Cardiovascular Drugs and Therapy (2022) Vol. 38, Iss. 2, pp. 253-262
Closed Access | Times Cited: 3
Inappropriate Activation of TLR4/NF-κB is a Cause of Heart Failure
Jiedong Zhou, Hui Lin, Tingting Lv, et al.
Cardiovascular Innovations and Applications (2022) Vol. 7, Iss. 1
Open Access | Times Cited: 3
Systems biology-based analysis exploring shared biomarkers and pathogenesis of myocardial infarction combined with osteoarthritis
Yuan Luo, Yongrui Liu, Weiqi Xue, et al.
Frontiers in Immunology (2024) Vol. 15
Open Access
Electroacupuncture alleviates myocardial ischemia–reperfusion injury by inhibiting hypothalamic paraventricular nucleus neurons projecting to the rostral ventrolateral medulla
Qi Shu, Jie Zhou, Bin Zhang, et al.
European Journal of Neuroscience (2024) Vol. 60, Iss. 5, pp. 4861-4876
Closed Access
Electroacupuncture alleviates MIRI by regulating hypothalamic paraventricular nucleus neurons projecting to the rostral ventrolateral medulla
Qi Shu, Jie Zhou, Bin Zhang, et al.
Authorea (Authorea) (2023)
Open Access
Implications for neutrophils in cardiac arrhythmias
Niklas Hegemann, Lukas Barth, Yannic Döring, et al.
AJP Heart and Circulatory Physiology (2023) Vol. 326, Iss. 3, pp. H441-H458
Open Access
Efeito do hormônio tiroideano na função cardíaca após injúria de isquemia-reperfusão em camundongos com bloqueio específico de NF-kB no cardiomiócito.
Denival Nascimento Vieira Júnior
(2023)
Open Access
Sigma-1R overexpression in PVN deteriorated heart injury by upregulating sympathetic nerve tone
Rui Wu, tingting Yu, xianglin Chen, et al.
Research Square (Research Square) (2022)
Open Access
Page 1

Scroll to top