OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Allosterism in the adenosine A2A and cannabinoid CB2 heteromer
Claudia Llinás del Torrent, Iu Raïch, Ángel González, et al.
British Journal of Pharmacology (2024)
Open Access | Times Cited: 6

Showing 6 citing articles:

Homodimerization of CB2 cannabinoid receptor triggered by a bivalent ligand enhances cellular signaling
Gemma Navarro, Marc Gómez‐Autet, Paula Morales, et al.
Pharmacological Research (2024) Vol. 208, pp. 107363-107363
Open Access | Times Cited: 15

Two-Step Cell Death Induction by the New 2-Arachidonoyl Glycerol Analog and Its Modulation by Lysophosphatidylinositol in Human Breast Cancer Cells
Mikhail Akimov, N. M. Gretskaya, Evgenia I. Gorbacheva, et al.
International Journal of Molecular Sciences (2025) Vol. 26, Iss. 2, pp. 820-820
Open Access

Attributes novel drug candidate: Constitutive GPCR signal bias mediated by purinergic receptors
Li Yin, Kexin Ni, Tianqi Mao, et al.
Pharmacology & Therapeutics (2025), pp. 108802-108802
Closed Access

Design of Small Non-Peptidic Ligands That Alter Heteromerization between Cannabinoid CB1 and Serotonin 5HT2A Receptors
Minos‐Timotheos Matsoukas, Marc Ciruela-Jardí, Maria Gallo, et al.
Journal of Medicinal Chemistry (2024)
Open Access

Homodimerization of CB2cannabinoid receptor triggered by a bivalent ligand enhances cellular signaling
Gemma Navarro, Marc Gómez‐Autet, Paula Morales, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access

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