OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

LncRNA HOTTIP promotes inflammatory response in acute gouty arthritis via miR‐101‐3p/BRD4 axis
Ping Shao, Huijie Liu, Yan-Yan Xue, et al.
International Journal of Rheumatic Diseases (2022) Vol. 26, Iss. 2, pp. 305-315
Closed Access | Times Cited: 11

Showing 11 citing articles:

Sepsis Biomarkers: Advancements and Clinical Applications—A Narrative Review
Rong‐Rong He, Guo-Li Yue, Meiling Dong, et al.
International Journal of Molecular Sciences (2024) Vol. 25, Iss. 16, pp. 9010-9010
Open Access | Times Cited: 15

Identification of Key lncRNAs in Gout Under Copper Death and Iron Death Mechanisms: A Study Based on ceRNA Network Analysis and Random Forest Algorithm
Zichen Shao, Weikang Sun, Qinqin Deng, et al.
Molecular Biotechnology (2024)
Closed Access | Times Cited: 4

BRD4 promotes gouty arthritis through MDM2-mediated PPARγ degradation and pyroptosis
Xiaoxia Xu, Hongbin Qiu
Molecular Medicine (2024) Vol. 30, Iss. 1
Open Access | Times Cited: 2

Long non-coding RNA HOTTIP induces inflammation in asthma by promoting EFNA3 transcription by CCCTC-binding factor.
Qiong Wu, Jinming Liu, Jie Deng, et al.
PubMed (2022) Vol. 14, Iss. 12, pp. 8903-8917
Closed Access | Times Cited: 4

LncRNA HOTTIP promotes LPS‐induced lung epithelial cell injury by recruiting DNMT1 to epigenetically regulate SP‐C
Shuang Li, Shuangjia Li, Zhanqun Gao, et al.
Journal of Cell Communication and Signaling (2024) Vol. 18, Iss. 1
Open Access

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