OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Epigenetic regulation of vascular smooth muscle cell phenotypic switch and neointimal formation by PRMT5
Ni Zhu, Zhi-Fu Guo, Kyosuke Kazama, et al.
Cardiovascular Research (2023) Vol. 119, Iss. 12, pp. 2244-2255
Open Access | Times Cited: 15

Showing 15 citing articles:

Vascular remodelling in cardiovascular diseases: hypertension, oxidation, and inflammation
Justyna Totoń‐Żurańska, T. Mikołajczyk, Blessy Saju, et al.
Clinical Science (2024) Vol. 138, Iss. 13, pp. 817-850
Closed Access | Times Cited: 27

Roles and mechanisms of histone methylation in vascular aging and related diseases
Yufei Ji, Zhenzhen Chen, Jun Cai
Clinical Epigenetics (2025) Vol. 17, Iss. 1
Open Access

Unraveling the metabolic‒epigenetic nexus: a new frontier in cardiovascular disease treatment
Jun Ouyang, Dong-de Wu, Y. Gan, et al.
Cell Death and Disease (2025) Vol. 16, Iss. 1
Open Access

The epigenetic regulation of crosstalk between cardiac fibroblasts and other cardiac cell types during stress
Lindsay Kraus, Salim Fredericks, Katelyn Scheeler
Frontiers in Cardiovascular Medicine (2025) Vol. 12
Open Access

SWI/SNF Complex in Vascular Smooth Muscle Cells and Its Implications in Cardiovascular Pathologies
Hongyu Liu, Yang Zhao, Guizhen Zhao, et al.
Cells (2024) Vol. 13, Iss. 2, pp. 168-168
Open Access | Times Cited: 3

Enhanced vasorin signaling mitigates adverse cardiovascular remodeling
Mingyi Wang, Kimberly R. McGraw, Robert E. Monticone, et al.
Aging Medicine (2024) Vol. 7, Iss. 3, pp. 414-423
Open Access | Times Cited: 2

Tudor-SN exacerbates pathological vascular remodeling by promoting the polyubiquitination of PTEN via NEDD4-1
Yichen Wu, Zilong Chen, Zhe Zheng, et al.
Journal of Biomedical Science (2024) Vol. 31, Iss. 1
Open Access | Times Cited: 2

Epigenetic regulation of vascular smooth muscle cell phenotypes in atherosclerosis
Jordi Lambert, Helle F. Jørgensen
Atherosclerosis (2024), pp. 119085-119085
Open Access

PF-477736 modulates vascular smooth muscle cells phenotypic transition through Chk1/p53/CD44 pathway
Yu Lv, Xia Wang, Youjie Zeng, et al.
Tissue and Cell (2024) Vol. 93, pp. 102682-102682
Closed Access

The epigenetic legacy of renin–angiotensin system inhibition in preventing hypertension
Ryszard Nosalski, Matteo Lemoli
Cardiovascular Research (2024) Vol. 120, Iss. 7, pp. 675-677
Open Access

Niclosamide modulates phenotypic switch and inflammatory responses in human pulmonary arterial smooth muscle cells
Yuwen Lu, Xiaogan Liang, Jingwen Song, et al.
Molecular and Cellular Biochemistry (2024)
Closed Access

Protein Arginine Methyltransferases: Emerging Targets in Cardiovascular and Metabolic Disease
Yan Zhang, Shibo Wei, Eun‐Ju Jin, et al.
Diabetes & Metabolism Journal (2024) Vol. 48, Iss. 4, pp. 487-502
Open Access

ZBED3 exacerbates hyperglycemia by promoting hepatic gluconeogenesis through CREB signaling
Yuanyuan Luo, Changshun Ruan, Fazhan Zhao, et al.
Metabolism (2024), pp. 156049-156049
Closed Access

Metabolites and Metabolism in Vascular Calcification: Links Between Adenosine Signaling and the Methionine Cycle
Parya Behzadi, Cynthia St. Hilaire
AJP Heart and Circulatory Physiology (2024) Vol. 327, Iss. 6, pp. H1361-H1375
Closed Access

Modulating the phenotypic transition of vascular smooth muscle cells via LKB1, a new pharmacologic target to strike atherosclerosis?
Luciano Consuegra‐Sánchez, Antonio Esteban-Luque, Juan Carlos Kaski
International Journal of Cardiology (2023) Vol. 395, pp. 131427-131427
Closed Access

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