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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

A novel epileptic encephalopathy mutation in KCNB1 disrupts Kv2.1 ion selectivity, expression, and localization
Isabelle Thiffault, David J. Speca, Daniel C. Austin, et al.
The Journal of General Physiology (2015) Vol. 146, Iss. 5, pp. 399-410
Open Access | Times Cited: 86

Showing 1-25 of 86 citing articles:

Ion Channels in Genetic Epilepsy: From Genes and Mechanisms to Disease-Targeted Therapies
Julia Oyrer, Snezana Maljevic, Ingrid E. Scheffer, et al.
Pharmacological Reviews (2017) Vol. 70, Iss. 1, pp. 142-173
Open Access | Times Cited: 266
Kv2 potassium channels form endoplasmic reticulum/plasma membrane junctions via interaction with VAPA and VAPB
Ben Johnson, Ashley N. Leek, Laura Solé, et al.
Proceedings of the National Academy of Sciences (2018) Vol. 115, Iss. 31
Open Access | Times Cited: 162
Developmental and epileptic encephalopathies: from genetic heterogeneity to phenotypic continuum
Renzo Guerrini, Valerio Conti, Massimo Mantegazza, et al.
Physiological Reviews (2022) Vol. 103, Iss. 1, pp. 433-513
Open Access | Times Cited: 91
Potassium Channel Gain of Function in Epilepsy: An Unresolved Paradox
Zachary Niday, Anastasios V. Tzingounis
The Neuroscientist (2018) Vol. 24, Iss. 4, pp. 368-380
Open Access | Times Cited: 130
Ion Channel Genes and Epilepsy: Functional Alteration, Pathogenic Potential, and Mechanism of Epilepsy
Feng Wei, Limin Yan, Tao Su, et al.
Neuroscience Bulletin (2017) Vol. 33, Iss. 4, pp. 455-477
Open Access | Times Cited: 123
Identification of VAPA and VAPB as Kv2 Channel-Interacting Proteins Defining Endoplasmic Reticulum–Plasma Membrane Junctions in Mammalian Brain Neurons
Michael Kirmiz, Nicholas C. Vierra, Stephanie Palacio, et al.
Journal of Neuroscience (2018) Vol. 38, Iss. 35, pp. 7562-7584
Open Access | Times Cited: 119
Neurodevelopmental Disorders Caused by De Novo Variants in KCNB1 Genotypes and Phenotypes
Carolien G. F. de Kovel, Steffen Syrbe, Eva H. Brilstra, et al.
JAMA Neurology (2017) Vol. 74, Iss. 10, pp. 1228-1228
Open Access | Times Cited: 89
Inactivation of the Kv2.1 channel through electromechanical coupling
Ana I. Fernández-Mariño, Xiao-Feng Tan, Chanhyung Bae, et al.
Nature (2023) Vol. 622, Iss. 7982, pp. 410-417
Open Access | Times Cited: 34
De novo KCNB1 mutations in infantile epilepsy inhibit repetitive neuronal firing
Hirotomo Saitsu, Tenpei Akita, Jun Tohyama, et al.
Scientific Reports (2015) Vol. 5, Iss. 1
Open Access | Times Cited: 80
Venom-derived peptide inhibitors of voltage-gated potassium channels
Raymond S. Norton, K. George Chandy
Neuropharmacology (2017) Vol. 127, pp. 124-138
Closed Access | Times Cited: 70
Remodeling neuronal ER–PM junctions is a conserved nonconducting function of Kv2 plasma membrane ion channels
Michael Kirmiz, Stephanie Palacio, Parashar Thapa, et al.
Molecular Biology of the Cell (2018) Vol. 29, Iss. 20, pp. 2410-2432
Open Access | Times Cited: 65
Spectrum of KV2.1 Dysfunction in KCNB1‐Associated Neurodevelopmental Disorders
Seok Kyu Kang, Carlos G. Vanoye, Sunita N. Misra, et al.
Annals of Neurology (2019) Vol. 86, Iss. 6, pp. 899-912
Open Access | Times Cited: 59
Clinical features and outcome of 6 new patients carrying de novo KCNB1 gene mutations
Carla Marini, Michele Romoli, Elena Parrini, et al.
Neurology Genetics (2017) Vol. 3, Iss. 6
Open Access | Times Cited: 59
Trafficking of Kv2.1 Channels to the Axon Initial Segment by a Novel Nonconventional Secretory Pathway
Camilla Stampe Jensen, Shôji Watanabe, Jeroen I. Stas, et al.
Journal of Neuroscience (2017) Vol. 37, Iss. 48, pp. 11523-11536
Open Access | Times Cited: 55
Ion Channels of the Islets in Type 2 Diabetes
David A. Jacobson, Show‐Ling Shyng
Journal of Molecular Biology (2019) Vol. 432, Iss. 5, pp. 1326-1346
Open Access | Times Cited: 48
Expanding the genetic and phenotypic relevance of KCNB1 variants in developmental and epileptic encephalopathies: 27 new patients and overview of the literature
Claire Bar, Giulia Barcia, Mélanie Jennesson, et al.
Human Mutation (2019) Vol. 41, Iss. 1, pp. 69-80
Open Access | Times Cited: 46
Intellectual Disability and Potassium Channelopathies: A Systematic Review
Miriam Kessi, Baiyu Chen, Jing Peng, et al.
Frontiers in Genetics (2020) Vol. 11
Open Access | Times Cited: 41
Potassium channels in behavioral brain disorders. Molecular mechanisms and therapeutic potential: A narrative review
Kazi Asraful Alam, Pernille Svalastoga, Aurora Martı́nez, et al.
Neuroscience & Biobehavioral Reviews (2023) Vol. 152, pp. 105301-105301
Open Access | Times Cited: 14
Ion channel traffic jams: the significance of trafficking deficiency in long QT syndrome
Gema Mondéjar‐Parreño, Ana I. Moreno-Manuel, Juan Manuel Ruiz-Robles, et al.
Cell Discovery (2025) Vol. 11, Iss. 1
Open Access
The Shab family potassium channels are highly enriched at the presynaptic terminals of human neurons
Orion Benner, Charles H. Karr, Astrid Quintero-Gonzalez, et al.
Journal of Biological Chemistry (2025), pp. 108235-108235
Open Access
Monogenic disorders that mimic the phenotype of Rett syndrome
Siddharth Srivastava, Sonal Desai, Julie S. Cohen, et al.
Neurogenetics (2018) Vol. 19, Iss. 1, pp. 41-47
Open Access | Times Cited: 46
Models for discovery of targeted therapy in genetic epileptic encephalopathies
Snezana Maljevic, Christopher A. Reid, Steven Petrou
Journal of Neurochemistry (2017) Vol. 143, Iss. 1, pp. 30-48
Open Access | Times Cited: 43
Kv2 channels create endoplasmic reticulum / plasma membrane junctions: a brief history of Kv2 channel subcellular localization
Ben Johnson, Ashley N. Leek, Michael M. Tamkun
Channels (2019) Vol. 13, Iss. 1, pp. 88-101
Open Access | Times Cited: 39
Mono-allelic KCNB2 variants lead to a neurodevelopmental syndrome caused by altered channel inactivation
Shreyas Bhat, Justine Rousseau, Coralie Michaud, et al.
The American Journal of Human Genetics (2024) Vol. 111, Iss. 4, pp. 761-777
Open Access | Times Cited: 4
A forward genetic screen identifies potassium channel essentiality in SHH medulloblastoma maintenance
Jerry J. Fan, Anders W. Erickson, Julia Carrillo-García, et al.
Developmental Cell (2025)
Closed Access
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