OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Loss of c-Cbl RING finger function results in high-intensity TCR signaling and thymic deletion
Christine B.F. Thien, Frøydis D. Blystad, Yifan Zhan, et al.
The EMBO Journal (2005) Vol. 24, Iss. 21, pp. 3807-3819
Open Access | Times Cited: 69

Showing 1-25 of 69 citing articles:

Frequent CBL mutations associated with 11q acquired uniparental disomy in myeloproliferative neoplasms
Francis Grand, Claire Hidalgo-Curtis, Thomas Ernst, et al.
Blood (2009) Vol. 113, Iss. 24, pp. 6182-6192
Open Access | Times Cited: 366

Protein tyrosine kinase regulation by ubiquitination: Critical roles of Cbl-family ubiquitin ligases
Bhopal Mohapatra, Gulzar Ahmad, Scott Nadeau, et al.
Biochimica et Biophysica Acta (BBA) - Molecular Cell Research (2012) Vol. 1833, Iss. 1, pp. 122-139
Open Access | Times Cited: 214

The Cbl family proteins: Ring leaders in regulation of cell signaling
Gayathri Swaminathan, Alexander Y. Tsygankov
Journal of Cellular Physiology (2006) Vol. 209, Iss. 1, pp. 21-43
Open Access | Times Cited: 285

Flt3-dependent transformation by inactivating c-Cbl mutations in AML
Bülent Sargin, Chunaram Choudhary, Nicola Crosetto, et al.
Blood (2007) Vol. 110, Iss. 3, pp. 1004-1012
Open Access | Times Cited: 207

Molecular mechanisms of CD4+ T-cell anergy
C. Garrison Fathman, Neil Lineberry
Nature reviews. Immunology (2007) Vol. 7, Iss. 8, pp. 599-609
Closed Access | Times Cited: 195

Degradation of Activated Protein Kinases by Ubiquitination
LU Zhi-min, Tony Hunter
Annual Review of Biochemistry (2009) Vol. 78, Iss. 1, pp. 435-475
Open Access | Times Cited: 134

Cbl and Human Myeloid Neoplasms: The Cbl Oncogene Comes of Age
Stephen C. Kales, Philip Ryan, Marion M. Nau, et al.
Cancer Research (2010) Vol. 70, Iss. 12, pp. 4789-4794
Open Access | Times Cited: 127

The E3 ubiquitin ligase c-Cbl restricts development and functions of hematopoietic stem cells
Chozhavendan Rathinam, Christine B.F. Thien, Wallace Y. Langdon, et al.
Genes & Development (2008) Vol. 22, Iss. 8, pp. 992-997
Open Access | Times Cited: 111

Myeloid Leukemia Development in c-Cbl RING Finger Mutant Mice Is Dependent on FLT3 Signaling
Chozhavendan Rathinam, Christine B.F. Thien, Richard A. Flavell, et al.
Cancer Cell (2010) Vol. 18, Iss. 4, pp. 341-352
Closed Access | Times Cited: 93

Essential Role of E3 Ubiquitin Ligase Activity in Cbl-b–Regulated T Cell Functions
Magdalena Paolino, Christine B.F. Thien, Thomas Gruber, et al.
The Journal of Immunology (2011) Vol. 186, Iss. 4, pp. 2138-2147
Open Access | Times Cited: 89

c-Cbl-Mediated Regulation of LAT-Nucleated Signaling Complexes
Lakshmi Balagopalan, Valarie A. Barr, Connie L. Sommers, et al.
Molecular and Cellular Biology (2007) Vol. 27, Iss. 24, pp. 8622-8636
Open Access | Times Cited: 100

CD45-Csk Phosphatase-Kinase Titration Uncouples Basal and Inducible T Cell Receptor Signaling during Thymic Development
Julie Zikherman, Craig N. Jenne, Susan R. Watson, et al.
Immunity (2010) Vol. 32, Iss. 3, pp. 342-354
Open Access | Times Cited: 84

Regulation of T‐cell tolerance by calcium/NFAT signaling
Ian Baine, Brian T. Abe, Fernando Macián
Immunological Reviews (2009) Vol. 231, Iss. 1, pp. 225-240
Closed Access | Times Cited: 84

CD5 dynamically calibrates basal NF-κB signaling in T cells during thymic development and peripheral activation
Courtney A. Matson, Seeyoung Choi, Ferenc Livák, et al.
Proceedings of the National Academy of Sciences (2020) Vol. 117, Iss. 25, pp. 14342-14353
Open Access | Times Cited: 44

Apoptosis during negative selection of autoreactive thymocytes
Seong-Wook Sohn, J THOMPSON, Aster Winoto
Current Opinion in Immunology (2007) Vol. 19, Iss. 5, pp. 510-515
Closed Access | Times Cited: 74

The why and how of thymocyte negative selection
Owen M. Siggs, Lydia Makaroff, Adrian Liston
Current Opinion in Immunology (2006) Vol. 18, Iss. 2, pp. 175-183
Open Access | Times Cited: 73

RING Finger E3 Ubiquitin Ligases: Structure and Drug Discovery
Christos T. Chasapis, Georgios A. Spyroulias
Current Pharmaceutical Design (2009) Vol. 15, Iss. 31, pp. 3716-3731
Closed Access | Times Cited: 58

Crk adaptor proteins mediate actin-dependent T cell migration and mechanosensing induced by the integrin LFA-1
Nathan H. Roy, Joanna L. MacKay, Tanner F. Robertson, et al.
Science Signaling (2018) Vol. 11, Iss. 560
Open Access | Times Cited: 45

Cell Regulation by Phosphotyrosine-Targeted Ubiquitin Ligases
Jonathan A. Cooper, Tomonori Kaneko, Shawn S.‐C. Li
Molecular and Cellular Biology (2015) Vol. 35, Iss. 11, pp. 1886-1897
Open Access | Times Cited: 43

LFA-1 signals to promote actin polymerization and upstream migration in T cells
Nathan H. Roy, Sarah Hyun Ji Kim, Alexander Buffone, et al.
Journal of Cell Science (2020) Vol. 133, Iss. 17
Open Access | Times Cited: 39

CBL mutations drive PI3K/AKT signaling via increased interaction with LYN and PIK3R1
Roger Belizaire, Sebastian Koochaki, Namrata D. Udeshi, et al.
Blood (2021) Vol. 137, Iss. 16, pp. 2209-2220
Open Access | Times Cited: 30

Rapidly fatal myeloproliferative disorders in mice with deletion of Casitas B-cell lymphoma (Cbl) and Cbl-b in hematopoietic stem cells
Mayumi Naramura, Neha Nandwani, Hua Gu, et al.
Proceedings of the National Academy of Sciences (2010) Vol. 107, Iss. 37, pp. 16274-16279
Open Access | Times Cited: 51

Mutant Cbl proteins as oncogenic drivers in myeloproliferative disorders
Mayumi Naramura, Scott Nadeau, Bhopal Mohapatra, et al.
Oncotarget (2011) Vol. 2, Iss. 3, pp. 245-250
Open Access | Times Cited: 47

Flt3 inhibitor AC220 is a potent therapy in a mouse model of myeloproliferative disease driven by enhanced wild-type Flt3 signaling
Samuel J. Taylor, Samantha A. Dagger, Christine B.F. Thien, et al.
Blood (2012) Vol. 120, Iss. 19, pp. 4049-4057
Open Access | Times Cited: 39

Human IgG1 antibodies suppress angiogenesis in a target-independent manner
Sasha Bogdanovich, Young‐Hee Kim, Takeshi Mizutani, et al.
Signal Transduction and Targeted Therapy (2016) Vol. 1, Iss. 1
Open Access | Times Cited: 35

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