OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

LET-381/FoxF and its target UNC-30/Pitx2 specify and maintain the molecular identity of C. elegans mesodermal glia that regulate motor behavior
Nikolaos Stefanakis, Jessica Jiang, Yupu Liang, et al.
The EMBO Journal (2024) Vol. 43, Iss. 6, pp. 956-992
Open Access | Times Cited: 6

Showing 6 citing articles:

Neurogenesis in Caenorhabditis elegans
Richard J. Poole, Nuria Flames, Luisa Cochella
Genetics (2024) Vol. 228, Iss. 2
Open Access | Times Cited: 3

UNC-30/PITX coordinates neurotransmitter identity with postsynaptic GABA receptor clustering
Edgar Correa, Morgane Mialon, Mélissa Cizeron, et al.
Development (2024) Vol. 151, Iss. 16
Open Access | Times Cited: 2

Tools and methods for cell ablation and cell inhibition in Caenorhabditis elegans
Dennis Rentsch, Amelie Bergs, Jiajie Shao, et al.
Genetics (2024)
Closed Access | Times Cited: 1

Glia in Invertebrate Models: Insights from Caenorhabditis elegans
Maria D. Purice, Liza J. Severs, Aakanksha Singhvi
Advances in neurobiology (2024), pp. 19-49
Closed Access | Times Cited: 1

Molecular profiling of invertebrate glia
Maria D. Purice, Inês Lago‐Baldaia, Vilaiwan M. Fernandes, et al.
Glia (2024) Vol. 73, Iss. 3, pp. 632-656
Open Access | Times Cited: 1

C. elegansLET-381/FoxF and DMD-4/DMRT control development of the mesodermal HMC endothelial cell
Nikolaos Stefanakis, Jasmine Xi, Jessica Jiang, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Closed Access

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