OpenAlex Citation Counts

OpenAlex Citations Logo

OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Endogenous opioids contribute to insensitivity to pain in humans and mice lacking sodium channel Nav1.7
Michael S. Minett, Vanessa Pereira, Shafaq Sikandar, et al.
Nature Communications (2015) Vol. 6, Iss. 1
Open Access | Times Cited: 176

Showing 1-25 of 176 citing articles:

The Role of Voltage-Gated Sodium Channels in Pain Signaling
David Bennett, Alex Clark, Jianying Huang, et al.
Physiological Reviews (2019) Vol. 99, Iss. 2, pp. 1079-1151
Open Access | Times Cited: 555

Persistent Postsurgical Pain
Philippe Richebé, Xavier Capdevila, Cyril Rivat
Anesthesiology (2018) Vol. 129, Iss. 3, pp. 590-607
Closed Access | Times Cited: 344

Breaking barriers to novel analgesic drug development
Ajay Yekkirala, David P. Roberson, Bruce P. Bean, et al.
Nature Reviews Drug Discovery (2017) Vol. 16, Iss. 8, pp. 545-564
Open Access | Times Cited: 313

Advances in understanding nociception and neuropathic pain
Ewan St. John Smith
Journal of Neurology (2017) Vol. 265, Iss. 2, pp. 231-238
Open Access | Times Cited: 238

Nav1.7 and other voltage-gated sodium channels as drug targets for pain relief
Edward C. Emery, Ana Paula Luiz, John N. Wood
Expert Opinion on Therapeutic Targets (2016) Vol. 20, Iss. 8, pp. 975-983
Open Access | Times Cited: 198

Defining the Functional Role of NaV1.7 in Human Nociception
Lucy McDermott, Greg A. Weir, Andreas C. Themistocleous, et al.
Neuron (2019) Vol. 101, Iss. 5, pp. 905-919.e8
Open Access | Times Cited: 171

Antibodies and venom peptides: new modalities for ion channels
Heike Wulff, Palle Christophersen, Paul A. Colussi, et al.
Nature Reviews Drug Discovery (2019) Vol. 18, Iss. 5, pp. 339-357
Open Access | Times Cited: 153

The anatomy of pain and suffering in the brain and its clinical implications
Dirk De Ridder, Divya Bharatkumar Adhia, Sven Vanneste
Neuroscience & Biobehavioral Reviews (2021) Vol. 130, pp. 125-146
Open Access | Times Cited: 152

The physiological function of different voltage-gated sodium channels in pain
George L. Goodwin, Stephen B. McMahon
Nature reviews. Neuroscience (2021) Vol. 22, Iss. 5, pp. 263-274
Closed Access | Times Cited: 112

Voltage-gated sodium channels in excitable cells as drug targets
Matthew Alsaloum, Sulayman D. Dib‐Hajj, Dana A. Page, et al.
Nature Reviews Drug Discovery (2025)
Closed Access | Times Cited: 3

Brain-derived neurotrophic factor derived from sensory neurons plays a critical role in chronic pain
Shafaq Sikandar, Michael S. Minett, Queensta Millet, et al.
Brain (2018) Vol. 141, Iss. 4, pp. 1028-1039
Open Access | Times Cited: 149

Nav1.7 and Nav1.8: Role in the pathophysiology of pain
Shaila Hameed
Molecular Pain (2019) Vol. 15
Open Access | Times Cited: 140

Pharmacological characterisation of the highly NaV1.7 selective spider venom peptide Pn3a
Jennifer R. Deuis, Zoltan Dekan, Joshua S. Wingerd, et al.
Scientific Reports (2017) Vol. 7, Iss. 1
Open Access | Times Cited: 131

NaV1.7 as a pain target – From gene to pharmacology
Irina Vetter, Jennifer R. Deuis, Alexander Mueller, et al.
Pharmacology & Therapeutics (2016) Vol. 172, pp. 73-100
Closed Access | Times Cited: 115

Insensitivity to pain induced by a potent selective closed-state Nav1.7 inhibitor
M.L. Flinspach, Q. Xu, Andrew D. Piekarz, et al.
Scientific Reports (2017) Vol. 7, Iss. 1
Open Access | Times Cited: 114

The role of Nav1.7 in human nociceptors: insights from human induced pluripotent stem cell–derived sensory neurons of erythromelalgia patients
Jannis Meents, Elisângela Bressan, Stephanie Sontag, et al.
Pain (2019) Vol. 160, Iss. 6, pp. 1327-1341
Open Access | Times Cited: 92

Gi-DREADD Expression in Peripheral Nerves Produces Ligand-Dependent Analgesia, as well as Ligand-Independent Functional Changes in Sensory Neurons
Jami L. Saloman, Nicole N. Scheff, Lindsey M. Snyder, et al.
Journal of Neuroscience (2016) Vol. 36, Iss. 42, pp. 10769-10781
Open Access | Times Cited: 91

Voltage-gated Na+ currents in human dorsal root ganglion neurons
Xiulin Zhang, Birgit T. Priest, Inna Belfer, et al.
eLife (2017) Vol. 6
Open Access | Times Cited: 90

The cellular and molecular basis of somatosensory neuron development
Shan Meltzer, Celine Santiago, Nikhil Sharma, et al.
Neuron (2021) Vol. 109, Iss. 23, pp. 3736-3757
Open Access | Times Cited: 90

Long-lasting analgesia via targeted in situ repression of Na V 1.7 in mice
Ana M. Moreno, Fernando Alemán, Glaucilene Ferreira Catroli, et al.
Science Translational Medicine (2021) Vol. 13, Iss. 584
Open Access | Times Cited: 83

A central mechanism of analgesia in mice and humans lacking the sodium channel NaV1.7
Donald Iain MacDonald, Shafaq Sikandar, Jan Weiss, et al.
Neuron (2021) Vol. 109, Iss. 9, pp. 1497-1512.e6
Open Access | Times Cited: 64

Neuropathic pain: From actual pharmacological treatments to new therapeutic horizons
Maxime Thouaye, İpek Yalçın
Pharmacology & Therapeutics (2023) Vol. 251, pp. 108546-108546
Open Access | Times Cited: 29

Nav1.7 is essential for nociceptor action potentials in the mouse in a manner independent of endogenous opioids
Lunbin Deng, Michelle Dourado, Rebecca M. Reese, et al.
Neuron (2023) Vol. 111, Iss. 17, pp. 2642-2659.e13
Open Access | Times Cited: 26

Page 1 - Next Page

Scroll to top