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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Peripheral Administration of a Cell-Penetrating MOTS-c Analogue Enhances Memory and Attenuates Aβ1–42- or LPS-Induced Memory Impairment through Inhibiting Neuroinflammation
Jinhong Jiang, Xin Chang, YaoYan Nie, et al.
ACS Chemical Neuroscience (2021) Vol. 12, Iss. 9, pp. 1506-1518
Closed Access | Times Cited: 27

Showing 1-25 of 27 citing articles:

Mitochondria-derived peptide MOTS-c: effects and mechanisms related to stress, metabolism and aging
Wei Wan, Lieliang Zhang, Yue Lin, et al.
Journal of Translational Medicine (2023) Vol. 21, Iss. 1
Open Access | Times Cited: 28
Danggui Shaoyao San: comprehensive modulation of the microbiota-gut-brain axis for attenuating Alzheimer’s disease-related pathology
Jiawei He, Yijie Jin, Chunxiang He, et al.
Frontiers in Pharmacology (2024) Vol. 14
Open Access | Times Cited: 13
Novel Insights into Mitochondrial DNA: Mitochondrial Microproteins and mtDNA Variants Modulate Athletic Performance and Age-Related Diseases
Hiroshi Kumagai, Brendan Miller, Su‐Jeong Kim, et al.
Genes (2023) Vol. 14, Iss. 2, pp. 286-286
Open Access | Times Cited: 19
The mitochondrial-derived peptide MOTS-c relieves hyperglycemia and insulin resistance in gestational diabetes mellitus
Yadong Yin, Yihui Pan, Jin He, et al.
Pharmacological Research (2021) Vol. 175, pp. 105987-105987
Open Access | Times Cited: 40
The DNA-dependent protein kinase catalytic subunit exacerbates endotoxemia-induced myocardial microvascular injury by disrupting the MOTS-c/JNK pathway and inducing profilin-mediated lamellipodia degradation
Rongjun Zou, Wanting Shi, Xing Chang, et al.
Theranostics (2024) Vol. 14, Iss. 4, pp. 1561-1582
Open Access | Times Cited: 5
MOTS-c, the Most Recent Mitochondrial Derived Peptide in Human Aging and Age-Related Diseases
Zahra Mohtashami, Mithalesh Kumar Singh, Nasim Salimiaghdam, et al.
International Journal of Molecular Sciences (2022) Vol. 23, Iss. 19, pp. 11991-11991
Open Access | Times Cited: 19
Dpep1 Mediates Regulation of Mitochondrial Quality Control Via Foxo1 to Attenuate Ferroptosis in Pulmonary Endothelial Cells to Attenuate Sali and the Protective Effect of the Mitochondria-Derived Peptide Mots-C
Xin Yao, Junjie Liao, Peng Lü, et al.
(2025)
Closed Access
The effect of gallic acid on memory and anxiety-like behaviors in rats with bile duct ligation-induced hepatic encephalopathy: Role of AMPK pathway.
Leila Jafaripour, Khadijeh Esmaeilpour, Marzieh Maneshian, et al.
DOAJ (DOAJ: Directory of Open Access Journals) (2022) Vol. 12, Iss. 4, pp. 425-438
Closed Access | Times Cited: 16
Mitochondrial-Derived Peptide MOTS-c Ameliorates Spared Nerve Injury-Induced Neuropathic Pain in Mice by Inhibiting Microglia Activation and Neuronal Oxidative Damage in the Spinal Cord via the AMPK Pathway
Jinhong Jiang, Lingfei Xu, Long Yang, et al.
ACS Chemical Neuroscience (2023) Vol. 14, Iss. 12, pp. 2362-2374
Closed Access | Times Cited: 9
The mitochondrial-derived peptide MOTS-c suppresses ferroptosis and alleviates acute lung injury induced by myocardial ischemia reperfusion via PPARγ signaling pathway
Peng Lü, Xiaopei Li, Ben Li, et al.
European Journal of Pharmacology (2023) Vol. 953, pp. 175835-175835
Open Access | Times Cited: 9
Current Advances in Mitochondrial Targeted Interventions in Alzheimer’s Disease
Tiago Siqueira de Sousa, Paula I. Moreira, Susana Cardoso
Biomedicines (2023) Vol. 11, Iss. 9, pp. 2331-2331
Open Access | Times Cited: 8
Adiponectin Attenuates Splenectomy-Induced Cognitive Deficits by Neuroinflammation and Oxidative Stress via TLR4/MyD88/NF-κb Signaling Pathway in Aged Rats
Zhi‐jing Zhang, Lideng Guo, Fei Yang, et al.
ACS Chemical Neuroscience (2023) Vol. 14, Iss. 10, pp. 1799-1809
Closed Access | Times Cited: 7
Orally administered MOTS-c analogue ameliorates dextran sulfate sodium-induced colitis by inhibiting inflammation and apoptosis
Jinhong Jiang, Xin Chang, YaoYan Nie, et al.
European Journal of Pharmacology (2022) Vol. 939, pp. 175469-175469
Closed Access | Times Cited: 12
The Mitochondrial-Derived Peptide (MOTS-c) Interacted with Nrf2 to Defend the Antioxidant System to Protect Dopaminergic Neurons Against Rotenone Exposure
Jingsong Xiao, Qifu Zhang, Yaohui Shan, et al.
Molecular Neurobiology (2023) Vol. 60, Iss. 10, pp. 5915-5930
Closed Access | Times Cited: 6
Mitochondria-derived peptides in healthy ageing and therapy of age-related diseases
Siarhei A. Dabravolski
Advances in protein chemistry and structural biology (2023), pp. 197-215
Closed Access | Times Cited: 5
Role of MOTS-c in the regulation of bone metabolism
Xuejie Yi, Guangxuan Hu, Yang� Yang, et al.
Frontiers in Physiology (2023) Vol. 14
Open Access | Times Cited: 4
Isoflurane impairs olfaction by increasing neuronal activity in the olfactory bulb
Hanyu Fu, Jingwei Zhou, Shan Li, et al.
Acta Physiologica (2023) Vol. 239, Iss. 1
Closed Access | Times Cited: 4
MOTS-c is an effective target for treating cancer-induced bone pain through the induction of AMPK-mediated mitochondrial biogenesis
Long Yang, Miaomiao Li, Yucheng Liu, et al.
Acta Biochimica et Biophysica Sinica (2024)
Open Access | Times Cited: 1
MOTS-c: A potential anti-pulmonary fibrosis factor derived by mitochondria
Zewei Zhang, Dongmei Chen, Kaili Du, et al.
Mitochondrion (2023) Vol. 71, pp. 76-82
Closed Access | Times Cited: 2
Central and peripheral mechanism of MOTS-c attenuates pain hypersensitivity in a mice model of inflammatory pain
Zhe Wang, Long Yang, Lingfei Xu, et al.
Neurological Research (2023) Vol. 46, Iss. 2, pp. 165-177
Closed Access | Times Cited: 2
MOTS-c is an effective target for treating streptozotocin induced painful diabetic neuropathy through induction of AMPK/PGC-1α -mediated mitochondrial biogenesis
Lingfei Xu, Xihui Tang, Long Yang, et al.
Molecular Pain (2024) Vol. 20
Open Access
Neuroprotective Mechanism of MOTS-c in TBI Mice: Insights from Integrated Transcriptomic and Metabolomic Analyses
Fengfeng Li, Jia Yang, Jun Fang, et al.
Drug Design Development and Therapy (2024) Vol. Volume 18, pp. 2971-2987
Open Access
Microproteins encoded by short open reading frames: vital regulators in neurological diseases
Xiao Xiao, Yitian Wang, Tingyu Li, et al.
Progress in Neurobiology (2024), pp. 102694-102694
Closed Access
Effects of intracerebroventricular MOTS-c infusion on thyroid hormones and uncoupling proteins
Mehmet Refik Bahar, Suat Tekin, A. Beytur, et al.
Biologia Futura (2023) Vol. 74, Iss. 1-2, pp. 159-170
Closed Access | Times Cited: 1
RETRACTED: Novel Insights Into the Role of Mitochondria-Derived Peptides in Myocardial Infarction
Dan Wu, Enny Kampmann, Geng Qian
Frontiers in Physiology (2021) Vol. 12
Open Access | Times Cited: 3
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