OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

SARS-CoV-2 ORF3a positively regulates NF-κB activity by enhancing IKKβ-NEMO interaction
Ying Nie, Lumin Mou, Qizhou Long, et al.
Virus Research (2023) Vol. 328, pp. 199086-199086
Open Access | Times Cited: 14

Showing 14 citing articles:

Mucosal immune response in biology, disease prevention and treatment
Xiaoxue Zhou, Yuchen Wu, Zhipeng Zhu, et al.
Signal Transduction and Targeted Therapy (2025) Vol. 10, Iss. 1
Open Access | Times Cited: 3

The role of SARS-CoV-2-mediated NF-κB activation in COVID-19 patients
Qiaoqiao Zhou, Lei Zhang, Yanming Dong, et al.
Hypertension Research (2023) Vol. 47, Iss. 2, pp. 375-384
Open Access | Times Cited: 20

Endoplasmic reticulum-associated SARS-CoV-2 ORF3a elicits heightened cytopathic effects despite robust ER-associated degradation
Jiantao Zhang, Ruth Cruz‐Cosme, Chen‐Yu Zhang, et al.
mBio (2023) Vol. 15, Iss. 1
Open Access | Times Cited: 13

Emerging regulatory mechanisms and functions of biomolecular condensates: implications for therapeutic targets
Soyoung Jeon, Yong‐Duck Chung, Jae‐Sung Lim, et al.
Signal Transduction and Targeted Therapy (2025) Vol. 10, Iss. 1
Open Access

A live attenuated SARS-CoV-2 vaccine constructed by dual inactivation of NSP16 and ORF3a
Zi‐Wei Ye, Chon Phin Ong, Hehe Cao, et al.
EBioMedicine (2025) Vol. 114, pp. 105662-105662
Closed Access

Impact of SARS-CoV-2 Wuhan and Omicron Variant Proteins on Type I Interferon Response
Marija Janevska, Evelien Naessens, Bruno Verhasselt
Viruses (2025) Vol. 17, Iss. 4, pp. 569-569
Open Access

Acute COVID-19 and LongCOVID syndrome – molecular implications for therapeutic strategies - review
Katarzyna Michalak, Alicja Michalak, Alicja Brenk-Krakowska
Frontiers in Immunology (2025) Vol. 16
Open Access

SARS-CoV-2 and the DNA damage response
Roger J.A. Grand
Journal of General Virology (2023) Vol. 104, Iss. 11
Open Access | Times Cited: 9

Linear ubiquitination mediates coronavirus NSP14-induced NF-κB activation
Fang Hua, Wenzhuo Hao, Lingyan Wang, et al.
Cell Communication and Signaling (2024) Vol. 22, Iss. 1
Open Access | Times Cited: 2

Potent Antiviral Activity of Vitamin B12 against Severe Acute Respiratory Syndrome Coronavirus 2, Middle East Respiratory Syndrome Coronavirus, and Human Coronavirus 229E
Yassmin Moatasim, Omnia Kutkat, Ahmed M. Osman, et al.
Microorganisms (2023) Vol. 11, Iss. 11, pp. 2777-2777
Open Access | Times Cited: 4

Longitudinal Assessment of Nasopharyngeal Biomarkers Post-COVID-19: Unveiling Persistent Markers and Severity Correlations
Francisco Javier Redondo Calvo, Yoana Rabanal‐Ruiz, Gema Verdugo-Moreno, et al.
Journal of Proteome Research (2024) Vol. 23, Iss. 11, pp. 5064-5084
Open Access | Times Cited: 1

SARS-CoV-2 ORF3a-Mediated NF-κB Activation Is Not Dependent on TRAF-Binding Sequence
Brianna M. Busscher, Henock B. Befekadu, Liu Z, et al.
Viruses (2023) Vol. 15, Iss. 11, pp. 2229-2229
Open Access | Times Cited: 3

Role of Transcription Factor NF-κB in Neuroimmunoendocrine Mechanisms of Respiratory Diseases
Yu. I. Belova, Ekaterina Mironova, T. S. Zubareva, et al.
Journal of Evolutionary Biochemistry and Physiology (2024) Vol. 60, Iss. 2, pp. 802-817
Closed Access

Transcription Factor NF-κB: Role and Significance in the Neuroimmunoendocrine Regulation of Respiratory Function in Normal Conditions and in Lung Pathology
Yu. I. Belova, Ekaterina Mironova, T. S. Zubareva, et al.
Российский физиологический журнал им  И  М  Сеченова (2024) Vol. 110, Iss. 4, pp. 527-546
Closed Access

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