OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Alogliptin exhibits multifaceted effects in thioacetamide-insulted rats: A novel approach to combating hepatic inflammation and fibrogenesis
Osama A. Mohammed
Pathology - Research and Practice (2023) Vol. 250, pp. 154833-154833
Closed Access | Times Cited: 4

Showing 4 citing articles:

Hedgehog signaling is a promising target for the treatment of hepatic fibrogenesis: a new management strategy using itraconazole-loaded nanoparticles
Elsayed A. Elmorsy, Sameh Saber, Ahmed Y. Kira, et al.
Frontiers in Pharmacology (2024) Vol. 15
Open Access | Times Cited: 5

Potential neuroprotective and autophagy-enhancing effects of alogliptin on lithium/pilocarpine-induced seizures in rats: Targeting the AMPK/SIRT1/Nrf2 axis
Sarah S El-Sayed, Shimaa O. Ali, Weam W. Ibrahim
Life Sciences (2024) Vol. 352, pp. 122917-122917
Closed Access | Times Cited: 3

Alogliptin attenuates testicular damage induced by monosodium glutamate in both juvenile and adult male rats by activating autophagy: ROS Dependent AMPK/mTOR
Manal Mohammad Morsy, Heba A. Hassan, Reham M. Morsi, et al.
Reproductive Toxicology (2024), pp. 108826-108826
Closed Access

Alogliptin attenuates STZ-induced diabetic nephropathy in rats through the modulation of autophagy, apoptosis, and inflammation pathways: Targeting NF-κB and AMPK/mTOR pathway
Salma Selim, Hassan M. El Fayoumi, Norhan M. El‐Sayed, et al.
Life Sciences (2024) Vol. 361, pp. 123307-123307
Closed Access

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