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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Hypoxic effects on the mitochondrial content and functions of the placenta in fetal growth restriction
Yue-Ying Xu, Yuan Liu, Ling Cui, et al.
Placenta (2021) Vol. 114, pp. 100-107
Closed Access | Times Cited: 22

Showing 22 citing articles:

Chlorpyrifos induces placental oxidative stress and barrier dysfunction by inducing mitochondrial apoptosis through the ERK/MAPK signaling pathway: In vitro and in vivo studies
Jun Bai, Siwei Deng, Huiyang Fu, et al.
The Science of The Total Environment (2023) Vol. 903, pp. 166449-166449
Closed Access | Times Cited: 16
Hypoxia-inducible factor 1 signaling drives placental aging and can provoke preterm labor
Erin J. Ciampa, Padraich Flahardy, Harini Srinivasan, et al.
eLife (2023) Vol. 12
Open Access | Times Cited: 13
Low Fetal Resistance to Hypoxia as a Cause of Stillbirth and Neonatal Encephalopathy
Петр Д. Шабанов, А. В. Самородов, Natalya Urakova, et al.
Clinical and Experimental Obstetrics & Gynecology (2024) Vol. 51, Iss. 2, pp. 33-33
Open Access | Times Cited: 4
Disparity Between Functional and Structural Recovery of Placental Mitochondria After Exposure to Hypoxia
Jonathan R. Sierla, Laia Pagerols Raluy, Magdalena Trochimiuk, et al.
International Journal of Molecular Sciences (2025) Vol. 26, Iss. 7, pp. 2956-2956
Open Access
The impact of early pregnancy metabolic disorders on pregnancy outcome and the specific mechanism
Xi-Zi Zhu, Zhimin Deng, Fangfang Dai, et al.
European journal of medical research (2023) Vol. 28, Iss. 1
Open Access | Times Cited: 10
Pain/Stress, Mitochondrial Dysfunction, and Neurodevelopment in Preterm Infants
Tingting Zhao, Xiaolin Chang, Subrata Kumar Biswas, et al.
Developmental Neuroscience (2024) Vol. 46, Iss. 5, pp. 341-352
Open Access | Times Cited: 3
Differential effect of lead and cadmium on mitochondrial function and NLRP3 inflammasome activation in human trophoblast
Yifeng Dai, Xijin Xu, Xia Huo, et al.
The Journal of Physiology (2024)
Open Access | Times Cited: 2
Mid-pregnancy placental transcriptome in a model of placental insufficiency with and without novel intervention
Rebecca L. Wilson, Baylea N. Davenport, Helen Jones
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access | Times Cited: 1
Extracellular vesicles as mediators of stress response in embryo-maternal communication
Seyed Omid Mousavi, Qurat Ul Ain Reshi, Kasun Godakumara, et al.
Frontiers in Cell and Developmental Biology (2024) Vol. 12
Open Access | Times Cited: 1
Mitochondrial Dysfunction and Oxidative Stress in Selective Fetal Growth Restriction
Yucheng Hu, Yuhong Lin, Jiawen Yang, et al.
Placenta (2024) Vol. 156, pp. 46-54
Open Access | Times Cited: 1
Maternal NO 2 exposure and fetal growth restriction: Hypoxia transmission and lncRNAs-proinflammation-mediated abnormal hematopoiesis
Liyao Qin, Huifeng Yue, Zhihua Gong, et al.
Proceedings of the National Academy of Sciences (2024) Vol. 121, Iss. 44
Closed Access | Times Cited: 1
Mitochondrial DNA quantification correlates with the developmental potential of human euploid blastocysts but not with that of mosaic blastocysts
Wen Luo, Yimin Zheng, Hao Yan, et al.
BMC Pregnancy and Childbirth (2023) Vol. 23, Iss. 1
Open Access | Times Cited: 3
Polysiloxane probes with main chain modifications regulating subcellular organelles localization for dynamic observation of lysosomal and mitochondrial interactions during cellular hypoxia
Ying Lan, Fanfan Wang, Yanfu Chai, et al.
Sensors and Actuators B Chemical (2023) Vol. 396, pp. 134646-134646
Closed Access | Times Cited: 2
The physiological basis with uterine myometrium contractions from electro-mechanical/hormonal myofibril function to the term and preterm labor
Farideh Zafari Zangeneh, Sedighe Hantoushzadeh
Heliyon (2023) Vol. 9, Iss. 11, pp. e22259-e22259
Open Access | Times Cited: 2
Effect of Hypoxia on Glucose Transporter 1 and 3 Gene Expression in Placental Mesenchymal Stem Cells Derived from Growth-Restricted Fetuses
Yao‐Lung Chang, Shuenn‐Dyh Chang, An‐Shine Chao, et al.
Genes (2022) Vol. 13, Iss. 5, pp. 752-752
Open Access | Times Cited: 4
Mid-Pregnancy Placental Transcriptome in a Model of Placental Insufficiency with and without Novel Intervention
Rebecca L. Wilson, Baylea N. Davenport, Helen Jones
Reproductive Sciences (2024)
Closed Access
Gestational exposure to carbon black nanoparticles triggered fetal growth restriction in mice: The mediation of inactivating autophagy-lysosomal degradation system in placental ferroptosis
Jing Li, Gao Hong-ying, Zehua Xu, et al.
The Science of The Total Environment (2024) Vol. 959, pp. 178167-178167
Closed Access
Hypoxia-inducible factor 1 signaling drives placental aging and can provoke preterm labor
Erin J. Ciampa, Padraich Flahardy, Harini Srinivasan, et al.
(2023)
Open Access | Times Cited: 1
Chlorpyrifos Induces Mitochondrial Apoptosis in Porcine Trophectoderm Cells Via Mapk Signaling Pathway
Jun Bai, Siwei Deng, Jun Li, et al.
SSRN Electronic Journal (2022)
Closed Access | Times Cited: 1
Hypoxia-inducible factor 1 signaling drives placental aging and can provoke preterm labor
Erin J. Ciampa, Padraich Flahardy, Harini Srinivasan, et al.
(2023)
Open Access
Hypoxia-inducible factor 1 signaling drives placental aging and can provoke preterm labor
Erin J. Ciampa, Padraich Flahardy, Harini Srinivasan, et al.
eLife (2023) Vol. 12
Open Access
Hypoxia-inducible factor 1 signaling drives placental aging and can provoke preterm labor
Erin J. Ciampa, Padraich Flahardy, Harini Srinivasan, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2022)
Open Access
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