OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Block or degrade? Balancing on- and off-target effects of antisense strategies against transcripts with expanded triplet repeats in DM1
Najoua El Boujnouni, M. Leontien van der Bent, Marieke Willemse, et al.
Molecular Therapy — Nucleic Acids (2023) Vol. 32, pp. 622-636
Open Access | Times Cited: 8

Showing 8 citing articles:

Antisense oligonucleotide therapeutic approach for Timothy syndrome
Xiaoyu Chen, Fikri Birey, Min-Yin Li, et al.
Nature (2024) Vol. 628, Iss. 8009, pp. 818-825
Open Access | Times Cited: 46

Mitigating off‐target effects of small RNAs: conventional approaches, network theory and artificial intelligence
Zoltán Bereczki, Bettina Benczik, Olivér M. Balogh, et al.
British Journal of Pharmacology (2024)
Open Access | Times Cited: 9

Therapeutic Antisense Oligonucleotides in Oncology: From Bench to Bedside
Elif Çakan, Olivia D. Lara, Anna Szymanowska, et al.
Cancers (2024) Vol. 16, Iss. 17, pp. 2940-2940
Open Access | Times Cited: 6

RNA and condensates: Disease implications and therapeutic opportunities
Tina W. Han, Bede Portz, Richard A. Young, et al.
Cell chemical biology (2024) Vol. 31, Iss. 9, pp. 1593-1609
Open Access | Times Cited: 4

CRISPR Diagnostics for Quantification and Rapid Diagnosis of Myotonic Dystrophy Type 1 Repeat Expansion Disorders
Kana Asano, Kazuto Yoshimi, Kohei Takeshita, et al.
ACS Synthetic Biology (2024) Vol. 13, Iss. 12, pp. 3926-3935
Open Access | Times Cited: 2

AntimiR treatment corrects myotonic dystrophy primary cell defects across several CTG repeat expansions with a dual mechanism of action
Estefanía Cerro-Herreros, Judit Núñez‐Manchón, Neia Naldaiz‐Gastesi, et al.
Science Advances (2024) Vol. 10, Iss. 41
Open Access

Systematic deletion of symmetrical CFTR exons reveals new therapeutic targets for exon skipping antisense oligonucleotides
C. Peña‐Rasgado, Elvia Rodriguez-Manriquez, Miroslav Dundr, et al.
Deleted Journal (2024) Vol. 1, Iss. 4
Open Access

All roads lead to cure: Diversity of oligonucleotides in DM1 therapy
Agnieszka Fiszer
Molecular Therapy — Nucleic Acids (2023) Vol. 32, pp. 898-899
Open Access | Times Cited: 1

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