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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Complement C3 mediates early hippocampal neurodegeneration and memory impairment in experimental multiple sclerosis
Julien Bourel, Vincent Planche, Nadège Dubourdieu, et al.
Neurobiology of Disease (2021) Vol. 160, pp. 105533-105533
Open Access | Times Cited: 41

Showing 1-25 of 41 citing articles:

Roles and regulation of microglia activity in multiple sclerosis: insights from animal models
Félix Distéfano‐Gagné, Sara Bitarafan, Steve Lacroix, et al.
Nature reviews. Neuroscience (2023) Vol. 24, Iss. 7, pp. 397-415
Closed Access | Times Cited: 54
Complement in the Brain: Contributions to Neuroprotection, Neuronal Plasticity, and Neuroinflammation
Suzanne S. Bohlson, Andrea J. Tenner
Annual Review of Immunology (2023) Vol. 41, Iss. 1, pp. 431-452
Open Access | Times Cited: 46
Complement C1q drives microglia-dependent synaptic loss and cognitive impairments in a mouse model of lipopolysaccharide-induced neuroinflammation
Xin-miao Wu, Yu-Zhu Gao, Cui-na Shi, et al.
Neuropharmacology (2023) Vol. 237, pp. 109646-109646
Closed Access | Times Cited: 32
Proximity Labeling Proteomics Reveals Kv1.3 Potassium Channel Immune Interactors in Microglia
Christine A Bowen, Hai M. Nguyen, Young Lin, et al.
Molecular & Cellular Proteomics (2024) Vol. 23, Iss. 8, pp. 100809-100809
Open Access | Times Cited: 8
Complement C1q-mediated microglial synaptic elimination by enhancing desialylation underlies sevoflurane-induced developmental neurotoxicity
Gang Wang, Huayue Liu, Xiaowen Meng, et al.
Cell & Bioscience (2024) Vol. 14, Iss. 1
Open Access | Times Cited: 6
Chronic experimental autoimmune encephalomyelitis is an excellent model to study neuroaxonal degeneration in multiple sclerosis
Rhonda R. Voskuhl, Allan MacKenzie‐Graham
Frontiers in Molecular Neuroscience (2022) Vol. 15
Open Access | Times Cited: 23
Peripheral inflammation as a potential mechanism and preventive strategy for perioperative neurocognitive disorder under general anesthesia and surgery
Yuan Li, Yingjie Li, Xu Fang, et al.
Frontiers in Cellular Neuroscience (2024) Vol. 18
Open Access | Times Cited: 5
Gypenoside XVII, an Active Ingredient from Gynostemma Pentaphyllum, Inhibits C3aR-Associated Synaptic Pruning in Stressed Mice
Man-Man Zhang, Guo-Ming Huo, Jie Cheng, et al.
Nutrients (2022) Vol. 14, Iss. 12, pp. 2418-2418
Open Access | Times Cited: 19
Instability of excitatory synapses in experimental autoimmune encephalomyelitis and the outcome for excitatory circuit inputs to individual cortical neurons
Rebecca L. Gillani, Eseza N. Kironde, Sara Whiteman, et al.
Brain Behavior and Immunity (2024) Vol. 119, pp. 251-260
Open Access | Times Cited: 4
Role of ginsenoside Rb1 in attenuating depression-like symptoms through astrocytic and microglial complement C3 pathway
Cheng‐Fu Li, Qiu-Ping Zhang, J. R. Cheng, et al.
Metabolic Brain Disease (2024) Vol. 39, Iss. 6, pp. 1039-1050
Closed Access | Times Cited: 4
Proteomic analysis reveals candidate molecules to mediate cortical pathology and identify possible biomarkers in an animal model of multiple sclerosis
Berenice Anabel Silva, María Celeste Leal, María Isabel Farías, et al.
Frontiers in Immunology (2025) Vol. 16
Open Access
Dual RNA-seq reveals the complement protein C3-mediated host-pathogen interaction in the brain abscess caused by Staphylococcus aureus
Qiyuan Jin, Yaxuan Zhai, Rui Qiang, et al.
mSystems (2025)
Open Access
Comparison of inflammatory biomarker levels in neurodegenerative proteinopathies: a case-control study
Sarah E. Cook, Kateřina Menšíková, Dorota Koníčková, et al.
Journal of Neural Transmission (2025)
Open Access
Astragalin-functionalized ultrasmall nanoparticles modulate the complement pathway to inhibit microglial synaptic phagocytosis for reducing anesthetic neurotoxicity
Gang Wang, Yaobao Han, Ke Peng, et al.
Materials Today Bio (2025) Vol. 32, pp. 101714-101714
Closed Access
Myeloid lineage C3 induces reactive gliosis and neuronal stress during CNS inflammation
Thomas Garton, Matthew D. Smith, Ajay Kesharwani, et al.
Nature Communications (2025) Vol. 16, Iss. 1
Open Access
Vulnerability of Thalamic Nuclei at CSF Interface During the Entire Course of Multiple Sclerosis
Ismail Koubiyr, Takayuki Yamamoto, Simon Blyau, et al.
Neurology Neuroimmunology & Neuroinflammation (2024) Vol. 11, Iss. 3
Open Access | Times Cited: 3
Free complement and complement containing extracellular vesicles as potential biomarkers for neuroinflammatory and neurodegenerative disorders
Marlies Burgelman, P Dujardin, Charysse Vandendriessche, et al.
Frontiers in Immunology (2023) Vol. 13
Open Access | Times Cited: 7
Viral-like TLR3 induction of cytokine networks and α-synuclein are reduced by complement C3 blockade in mouse brain
Ria Thomas, Kyle J. Connolly, Oeystein Roed Brekk, et al.
Scientific Reports (2023) Vol. 13, Iss. 1
Open Access | Times Cited: 7
Serum-Based Biomarkers in Neurodegeneration and Multiple Sclerosis
Patrizia LoPresti
Biomedicines (2022) Vol. 10, Iss. 5, pp. 1077-1077
Open Access | Times Cited: 11
Astrocyte-secreted C3 signaling impairs neuronal development and cognition in autoimmune diseases
Fan Zhu, Pengyan He, Wei Jiang, et al.
Progress in Neurobiology (2024) Vol. 240, pp. 102654-102654
Closed Access | Times Cited: 2
Complement 3a induces the synapse loss via C3aR in mitochondria-dependent NLRP3 activating mechanisms during the development and progression of Alzheimer’s disease
Tong-Qi Ge, Peipei Guan, Pu Wang
Neuroscience & Biobehavioral Reviews (2024) Vol. 165, pp. 105868-105868
Closed Access | Times Cited: 2
Complement factor B is essential for the proper function of the peripheral auditory system
LaShardai N. Brown, Jeremy L. Barth, Shabih H. Jafri, et al.
Frontiers in Neurology (2023) Vol. 14
Open Access | Times Cited: 5
The validity of animal models to explore the pathogenic role of the complement system in multiple sclerosis: A review
Nil Saez-Calveras, Amy L. Brewster, Olaf Stüve
Frontiers in Molecular Neuroscience (2022) Vol. 15
Open Access | Times Cited: 8
Complement and microglia activation mediate stress-induced synapse loss in layer 2/3 of the medial prefrontal cortex in male mice
Haven Tillmon, Breeanne M. Soteros, Liang Shen, et al.
Nature Communications (2024) Vol. 15, Iss. 1
Open Access | Times Cited: 1
Should We Consider Neurodegeneration by Itself or in a Triangulation with Neuroinflammation and Demyelination? The Example of Multiple Sclerosis and Beyond
Océane Perdaens, Vincent Van Pesch
International Journal of Molecular Sciences (2024) Vol. 25, Iss. 23, pp. 12637-12637
Open Access | Times Cited: 1
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