OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Synthesis of new sulfonamide derivatives: Investigation of their interactions with carbonic anhydrase and cholinesterase enzymes by in vitro and in silico evaluations
Muhammet Gürkan Kurban, Reşit Çakmak, Eyüp Başaran, et al.
Journal of Molecular Structure (2024) Vol. 1314, pp. 138798-138798
Closed Access | Times Cited: 4

Showing 4 citing articles:

Synthesis, biological evaluation, and in silico studies of phenyl naphthalene-2-sulfonate derived thiosemicarbazones as potential carbonic anhydrase inhibitors
Javeria Eshal, Hafiza Zara Tariq, Jing Li, et al.
Bioorganic Chemistry (2025) Vol. 155, pp. 108118-108118
Closed Access

Synthesis of new fluorinated sulfonates and their Schiff bases as anti-Alzheimer drug candidates: An in vitro-in silico study
Reşit Çakmak, Eyüp Başaran, Selami Ercan, et al.
Journal of Molecular Structure (2025), pp. 141474-141474
Closed Access

Synthetic strategies and medicinal chemistry perspectives of dual acting carbonic anhydrase modulators with monoamine oxidase and cholinesterase inhibitors
Sandeep Bindra, Ehab M. Mostafa, Mohamed A. Abdelgawad, et al.
RSC Medicinal Chemistry (2025)
Closed Access

Design, synthesis of antipyrine-based Schiff bases and investigation of their cholinesterase and carbonic anhydrase activities by in vitro and in silico approaches
Reşit Çakmak, Eyüp Başaran, Burçin Türkmenoğlu, et al.
Journal of Molecular Structure (2024), pp. 140599-140599
Closed Access

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