OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

p21 induces a senescence program and skeletal muscle dysfunction
Davis A. Englund, Alyssa M. Jolliffe, Zaira Aversa, et al.
Molecular Metabolism (2022) Vol. 67, pp. 101652-101652
Open Access | Times Cited: 50

Showing 1-25 of 50 citing articles:

Exercise metabolism and adaptation in skeletal muscle
Jonathon A.B. Smith, Kevin A. Murach, Kenneth A. Dyar, et al.
Nature Reviews Molecular Cell Biology (2023) Vol. 24, Iss. 9, pp. 607-632
Open Access | Times Cited: 111

Guidelines for minimal information on cellular senescence experimentation in vivo
Mikołaj Ogrodnik, Juan Carlos Acosta, Peter D. Adams, et al.
Cell (2024) Vol. 187, Iss. 16, pp. 4150-4175
Open Access | Times Cited: 64

Biomarkers of cellular senescence and risk of death in humans
Jennifer L. St. Sauver, Susan A. Weston, Elizabeth J. Atkinson, et al.
Aging Cell (2023) Vol. 22, Iss. 12
Open Access | Times Cited: 39

Cellular senescence in skeletal disease: mechanisms and treatment
Xu He, Wei Hu, Yuanshu Zhang, et al.
Cellular & Molecular Biology Letters (2023) Vol. 28, Iss. 1
Open Access | Times Cited: 34

Calorie restriction reduces biomarkers of cellular senescence in humans
Zaira Aversa, Thomas A. White, Amanda A. Heeren, et al.
Aging Cell (2023) Vol. 23, Iss. 2
Open Access | Times Cited: 28

The role of p21 in cellular senescence and aging-related diseases
Jiayu Yan, Siyi Chen, Zimei Yi, et al.
Molecules and Cells (2024), pp. 100113-100113
Open Access | Times Cited: 12

Biomarkers of Cellular Senescence Predict the Onset of Mobility Disability and Are Reduced by Physical Activity in Older Adults
Roger A. Fielding, Elizabeth J. Atkinson, Zaira Aversa, et al.
The Journals of Gerontology Series A (2023) Vol. 79, Iss. 3
Open Access | Times Cited: 17

Cellular Senescence and Inflammaging in the Bone: Pathways, Genetics, Anti-Aging Strategies and Interventions
Merin Lawrence, Abhishek Goyal, Shelly Pathak, et al.
International Journal of Molecular Sciences (2024) Vol. 25, Iss. 13, pp. 7411-7411
Open Access | Times Cited: 6

Senescent fibro-adipogenic progenitors are potential drivers of pathology in inclusion body myositis
Christopher Nelke, Christina B. Schroeter, Lukas Theissen, et al.
Acta Neuropathologica (2023) Vol. 146, Iss. 5, pp. 725-745
Open Access | Times Cited: 16

CDKN1A/p21 in Breast Cancer: Part of the Problem, or Part of the Solution?
Evangelos Manousakis, Clàudia Martinez Miralles, Maria Guimerà Esquerda, et al.
International Journal of Molecular Sciences (2023) Vol. 24, Iss. 24, pp. 17488-17488
Open Access | Times Cited: 16

Senotherapeutic drug treatment ameliorates chemotherapy-induced cachexia
Davis A. Englund, Alyssa M. Jolliffe, Gabriel J. Hanson, et al.
JCI Insight (2023) Vol. 9, Iss. 2
Open Access | Times Cited: 14

Effects of Hsa-miR-4741/LILRB2 on Senescence of Nucleus Pulposus Cells and Their Prognostic Values in Lumbar Disc Herniation
Ziwen He, Nanfeng Zheng, Xiu-Quan Guo, et al.
Journal of Investigative Surgery (2025) Vol. 38, Iss. 1
Open Access

Dnmt3a overexpression disrupts skeletal muscle homeostasis, promotes an aging-like phenotype, and reduces metabolic elasticity
Mamoru Oyabu, Yuto Ohira, Mariko Fujita, et al.
iScience (2025) Vol. 28, Iss. 4, pp. 112144-112144
Open Access

Propagation of senescent phenotypes by extracellular HMGB1 is dependent on its redox state
Jiwon Shin, Dong‐Hyun Jang, So Young Kim, et al.
Metabolism (2025), pp. 156259-156259
Closed Access

Human myofiber‐enriched aging‐induced lncRNAFRAIL1 promotes loss of skeletal muscle function
Matthew J. Miller, Kevin J. Gries, George R. Marcotte, et al.
Aging Cell (2024) Vol. 23, Iss. 4
Open Access | Times Cited: 3

Crosstalk between the DNA damage response and cellular senescence drives aging and age-related diseases
Ajmal Ahmad, Anneliesse Braden, Sazzad Khan, et al.
Seminars in Immunopathology (2024) Vol. 46, Iss. 3-4
Closed Access | Times Cited: 3

Cholesterol Accumulation Promotes Photoreceptor Senescence and Retinal Degeneration
Ryo Terao, Brian S. Sohn, Taku Yamamoto, et al.
Investigative Ophthalmology & Visual Science (2024) Vol. 65, Iss. 10, pp. 29-29
Open Access | Times Cited: 3

Cellular Senescence: The Driving Force of Musculoskeletal Diseases
Angela Falvino, Beatrice Gasperini, Ida Cariati, et al.
Biomedicines (2024) Vol. 12, Iss. 9, pp. 1948-1948
Open Access | Times Cited: 3

The life and times of cellular senescence in skeletal muscle: friend or foe for homeostasis and adaptation?
Cory M. Dungan, Jaden M. Wells, Kevin A. Murach
AJP Cell Physiology (2023) Vol. 325, Iss. 1, pp. C324-C331
Closed Access | Times Cited: 8

Cellular senescence and frailty: a comprehensive insight into the causal links
Serena Marcozzi, Giorgia Bigossi, Maria Giuliani, et al.
GeroScience (2023) Vol. 45, Iss. 6, pp. 3267-3305
Open Access | Times Cited: 7

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