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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Early alveolar epithelial cell necrosis is a potential driver of COVID-19-induced acute respiratory distress syndrome
Kentaro Tojo, Natsuhiro Yamamoto, Nao Tamada, et al.
iScience (2022) Vol. 26, Iss. 1, pp. 105748-105748
Open Access | Times Cited: 22

Showing 22 citing articles:

Cigarette Smoke-Induced Respiratory Response: Insights into Cellular Processes and Biomarkers
Sang-Ryul Cha, Jimin Jang, Sung‐Min Park, et al.
Antioxidants (2023) Vol. 12, Iss. 6, pp. 1210-1210
Open Access | Times Cited: 33
Ferroptosis and pyroptosis signatures in critical COVID-19 patients
Cédric Peleman, Samya Van Coillie, Symen Ligthart, et al.
Cell Death and Differentiation (2023) Vol. 30, Iss. 9, pp. 2066-2077
Open Access | Times Cited: 20
Renin-Angiotensin System and Sex Differences in COVID-19: A Critical Assessment
Mark C. Chappell
Circulation Research (2023) Vol. 132, Iss. 10, pp. 1320-1337
Open Access | Times Cited: 17
Identification of FasL as a crucial host factor driving COVID-19 pathology and lethality
Marie‐Christine Albert, Iratxe Uranga-Murillo, Maykel Arias, et al.
Cell Death and Differentiation (2024) Vol. 31, Iss. 5, pp. 544-557
Open Access | Times Cited: 5
Acute lung injury: a view from the perspective of necroptosis
Jinyan Dong, Weihong Liu, Wenli Liu, et al.
Inflammation Research (2024) Vol. 73, Iss. 6, pp. 997-1018
Closed Access | Times Cited: 5
Pathophysiological mechanisms of ARDS: a narrative review from molecular to organ-level perspectives
Kaihuan Zhou, Qianqian Qin, Junyu Lu
Respiratory Research (2025) Vol. 26, Iss. 1
Open Access
Identification of Early Biomarkers of Mortality in COVID-19 Hospitalized Patients: A LASSO-Based Cox and Logistic Approach
Anna Maria Fratta Pasini, Chiara Stranieri, Edoardo Giuseppe Di Leo, et al.
Viruses (2025) Vol. 17, Iss. 3, pp. 359-359
Open Access
Interleukin-6 regulates human ODAM gene expression in gingival epithelial cells
Zhenyu Jin, Arisa Yamaguchi, Hideki Takai, et al.
Journal of Periodontal & Implant Science (2025) Vol. 55
Closed Access
Ferroptosis: a novel mechanism of cell death in ophthalmic conditions
Yaqi Yang, Yumeng Lin, Zhongyu Han, et al.
Frontiers in Immunology (2024) Vol. 15
Open Access | Times Cited: 4
Alveolar epithelial cells mitigate neutrophilic inflammation in lung injury through regulating mitochondrial fatty acid oxidation
Kuei‐Pin Chung, Chih‐Ning Cheng, Yi‐Jung Chen, et al.
Nature Communications (2024) Vol. 15, Iss. 1
Open Access | Times Cited: 4
COVID-19 Biogenesis and Intracellular Transport
Alexander A. Mirоnоv, M.A. Savin, Galina V. Beznoussenko
International Journal of Molecular Sciences (2023) Vol. 24, Iss. 5, pp. 4523-4523
Open Access | Times Cited: 11
RAGE engagement by SARS-CoV-2 enables monocyte infection and underlies COVID-19 severity
Roberta Angioni, Matteo Bonfanti, Nicolò Caporale, et al.
Cell Reports Medicine (2023) Vol. 4, Iss. 11, pp. 101266-101266
Open Access | Times Cited: 10
Global prevalence of COVID-19-induced acute respiratory distress syndrome: systematic review and meta-analysis
Abere Woretaw Azagew, Zerko Wako Beko, Yohannes Mulu Ferede, et al.
Systematic Reviews (2023) Vol. 12, Iss. 1
Open Access | Times Cited: 10
Self-DNA driven inflammation in COVID-19 and after mRNA-based vaccination: lessons for non-COVID-19 pathologies
Martin Heil
Frontiers in Immunology (2024) Vol. 14
Open Access | Times Cited: 3
The Anti-Oxidative, Anti-Inflammatory, Anti-Apoptotic, and Anti-Necroptotic Role of Zinc in COVID-19 and Sepsis
George Briassoulis, Panagiotis Briassoulis, Stavroula Ilia, et al.
Antioxidants (2023) Vol. 12, Iss. 11, pp. 1942-1942
Open Access | Times Cited: 9
Necroptosis in alveolar epithelial cells drives lung inflammation and injury caused by SARS-CoV-2 infection
Yoji Komiya, Mari Kamiya, Seiya Oba, et al.
Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease (2024) Vol. 1870, Iss. 8, pp. 167472-167472
Closed Access | Times Cited: 1
COVID-19 PBMCs are doubly harmful, through LDN-mediated lung epithelial damage and monocytic impaired responsiveness to live Pseudomonas aeruginosa exposure
Clémence Gaudin, Maëlys Born-Bony, Bérengère Villeret, et al.
Frontiers in Immunology (2024) Vol. 15
Open Access
Building an Adverse Outcome Pathway network for COVID-19
Penny Nymark, Laure‐Alix Clerbaux, Maria João Amorim, et al.
Frontiers in Systems Biology (2024) Vol. 4
Open Access
Early inflammatory profiles predict maximal disease severity in COVID-19: An unsupervised cluster analysis
Grace Kenny, Gurvin Saini, Colette Marie Gaillard, et al.
Heliyon (2024) Vol. 10, Iss. 15, pp. e34694-e34694
Open Access
COVID-19 Lung Injury: Unique and Familiar Aspects of Pathophysiology
Stefan Hall, Geraint Berger, Christine Lehmann
Applied Sciences (2024) Vol. 14, Iss. 23, pp. 11048-11048
Open Access
Rhei Radix et Rhizoma in Xuanbai-Chengqi decoction strengthens the intestinal barrier function and promotes lung barrier repair in preventing severe viral pneumonia induced by influenza A virus
Lin-Feng Guo, Weilian Bao, Shuiyuan Yang, et al.
Journal of Ethnopharmacology (2023) Vol. 319, pp. 117231-117231
Closed Access | Times Cited: 1
Molecular basis of COVID-19, ARDS and COVID-19-associated ARDS: Diagnosis pathogenesis and therapeutic strategies
PRIYADHARSHINI THANJAVUR SRIRAMAMOORTHI, Gayathri Gopal, Shibi Muralidar, et al.
Biocell (2023) Vol. 47, Iss. 11, pp. 2335-2350
Open Access
Identifying Necroptosis Signature Genes Based on Machine Learning for Evaluating COVID-19 Progression
T. Feng, Yitong Li, Yue Wu, et al.
(2023), pp. 23-31
Closed Access
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