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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

iPSC motor neurons, but not other derived cell types, capture gene expression changes in postmortem sporadic ALS motor neurons
Aaron Held, Michelle Adler, Christine Marques, et al.
Cell Reports (2023) Vol. 42, Iss. 9, pp. 113046-113046
Open Access | Times Cited: 13

Showing 13 citing articles:

A model of human neural networks reveals NPTX2 pathology in ALS and FTLD
Marián Hruška-Plocháň, Vera I. Wiersma, A Betz, et al.
Nature (2024) Vol. 626, Iss. 8001, pp. 1073-1083
Open Access | Times Cited: 16
Neuronal STING activation in amyotrophic lateral sclerosis and frontotemporal dementia
Christine Marques, Aaron Held, Katherine Dorfman, et al.
Acta Neuropathologica (2024) Vol. 147, Iss. 1
Open Access | Times Cited: 15
SLC22A17 as a Cell Death–Linked Regulator of Tight Junctions in Cerebral Ischemia
Wenlu Li, Jingfei Shi, Zhanyang Yu, et al.
Stroke (2024) Vol. 55, Iss. 6, pp. 1650-1659
Closed Access | Times Cited: 5
ALS-Associated TDP-43 Dysfunction Compromises UPF1-Dependent mRNA Metabolism Pathways Including Alternative Polyadenylation and 3’UTR Length
Francesco Alessandrini, Matthew Wright, Tatsuaki Kurosaki, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access | Times Cited: 4
Decoding Neurodegeneration: A Review of Molecular Mechanisms and Therapeutic Advances in Alzheimer’s, Parkinson’s, and ALS
Corneliu Toader, Călin Petru Tătaru, Octavian Munteanu, et al.
International Journal of Molecular Sciences (2024) Vol. 25, Iss. 23, pp. 12613-12613
Open Access | Times Cited: 4
Mitochondrial respiratory complex IV deficiency recapitulates amyotrophic lateral sclerosis
Cheng Man, Dan Lü, Kaihui Li, et al.
Nature Neuroscience (2025)
Closed Access
Physiological regulation of neuronal Wnt activity is essential for TDP-43 localization and function
Nan Zhang, Anna Westerhaus, Macey Wilson, et al.
The EMBO Journal (2024) Vol. 43, Iss. 16, pp. 3388-3413
Open Access | Times Cited: 3
Highly variable molecular signatures of TDP-43 loss of function are associated with nuclear pore complex injury in a population study of sporadic ALS patient iPSNs
Jeffrey D. Rothstein, Caroline Warlick, Alyssa N. Coyne
bioRxiv (Cold Spring Harbor Laboratory) (2023)
Open Access | Times Cited: 8
Early disruption of the CREB pathway drives dendritic morphological alterations in FTD/ALS cortical neurons
Michelle Gregoire, Riccardo Sirtori, L Donatelli, et al.
Proceedings of the National Academy of Sciences (2024) Vol. 121, Iss. 49
Closed Access | Times Cited: 1
Counter-regulation of RNA stability by UPF1 and TDP43
Nicolás Gómez, Caroline Hsieh, Xingli Li, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access
Stathmin 2 is a potential treatment target for TDP-43 proteinopathy in amyotrophic lateral sclerosis
Yunqing Liu, Dejun Yan, Lin Yang, et al.
Translational Neurodegeneration (2024) Vol. 13, Iss. 1
Open Access
Cryptic exon inclusion in TDP-43 proteinopathies: opportunities and challenges
Lorena Decker, Sonja Menge, Axel Freischmidt
Neural Regeneration Research (2024) Vol. 20, Iss. 7, pp. 2003-2004
Open Access
Axonal transcriptome reveals upregulation of PLK1 as a protective mechanism in response to increased DNA damage in FUSP525L spinal motor neurons
Vitaly Zimyanin, Banaja P. Dash, Dajana Großmann, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access
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