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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

SNHG16 aggravates chronic constriction injury-induced neuropathic pain in rats via binding with miR-124-3p and miR-141-3p to upregulate JAG1
Hui Li, Lianlian Fan, Yan Zhang, et al.
Brain Research Bulletin (2020) Vol. 165, pp. 228-237
Closed Access | Times Cited: 23

Showing 23 citing articles:

Advances in the role and mechanism of miRNA in inflammatory pain
Xiaoyu Zhang, Lin Zhu, Xuezhen Wang, et al.
Biomedicine & Pharmacotherapy (2023) Vol. 161, pp. 114463-114463
Open Access | Times Cited: 15
Ursolic acid ameliorates traumatic brain injury in mice by regulating microRNA-141-mediated PDCD4/PI3K/AKT signaling pathway
Hongyun Zhang, Zhenyi Xing, Jie Zheng, et al.
International Immunopharmacology (2023) Vol. 120, pp. 110258-110258
Closed Access | Times Cited: 13
JAG1 enhances angiogenesis in triple-negative breast cancer through promoting the secretion of exosomal lncRNA MALAT1
Junping Liu, Yutong Shi, Minmin Wu, et al.
Genes & Diseases (2022) Vol. 10, Iss. 5, pp. 2167-2178
Open Access | Times Cited: 21
New Vistas in microRNA Regulatory Interactome in Neuropathic Pain
Yash Gada, Amitkumar Pandey, Nikita Jadhav, et al.
Frontiers in Pharmacology (2022) Vol. 12
Open Access | Times Cited: 19
LncRNA 4933431K23Rik modulate microglial phenotype via inhibiting miR-10a-5p in spinal cord injury induced neuropathic pain
Changxuan You, Waiping Zhou, Ping Ye, et al.
Scientific Reports (2025) Vol. 15, Iss. 1
Open Access
The potential role of miR-124-3p in tumorigenesis and other related diseases
Qian Li, Shuqing Liu, Jinsong Yan, et al.
Molecular Biology Reports (2021) Vol. 48, Iss. 4, pp. 3579-3591
Closed Access | Times Cited: 25
LncRNA CRNDE exacerbates neuropathic pain in chronic constriction injury-induced(CCI) rats through regulating miR-146a-5p/WNT5A pathway
Qiangze Zhang, Dongxia Zhu, Qiang Li
Bioengineered (2021) Vol. 12, Iss. 1, pp. 7348-7359
Open Access | Times Cited: 17
The role of non-coding RNAs in neuropathic pain
Xiuying He, Huisi Yang, Yuexiang Zheng, et al.
Pflügers Archiv - European Journal of Physiology (2024) Vol. 476, Iss. 11, pp. 1625-1643
Closed Access | Times Cited: 2
The Role of miRNAs in Neuropathic Pain
Martina Morchio, Emanuele Sher, David Collier, et al.
Biomedicines (2023) Vol. 11, Iss. 3, pp. 775-775
Open Access | Times Cited: 6
Transcriptome Profiling of miRNA-mRNA Interactions and Associated Mechanisms in Chemotherapy-Induced Neuropathic Pain
Xiao-Hua Yang, Xiqiang Huang, Weicheng Lu, et al.
Molecular Neurobiology (2023) Vol. 60, Iss. 10, pp. 5672-5690
Closed Access | Times Cited: 6
miR-141-3p inhibits the activation of astrocytes and the release of inflammatory cytokines in bacterial meningitis through down-regulating HMGB1
Xiao Fang, Huaili Wang, Zhihong Zhuo, et al.
Brain Research (2021) Vol. 1770, pp. 147611-147611
Closed Access | Times Cited: 14
Roles of Long Non-coding RNAs in the Development of Chronic Pain
Zheng Li, Xiongjuan Li, Wenling Jian, et al.
Frontiers in Molecular Neuroscience (2021) Vol. 14
Open Access | Times Cited: 14
Identification of Slc6a19os and SOX11 as Two Novel Essential Genes in Neuropathic Pain Using Integrated Bioinformatic Analysis and Experimental Verification
Peng Chen, Chen Wang, Dongsheng Lin, et al.
Frontiers in Neuroscience (2021) Vol. 15
Open Access | Times Cited: 13
LncRNA SNHG16 regulates trophoblast functions by the miR-218-5p/LASP1 axis
Yu Zhou, Yulei Zhang, Hao‐Yu Zheng, et al.
Journal of Molecular Histology (2021) Vol. 52, Iss. 5, pp. 1021-1033
Closed Access | Times Cited: 13
Propofol alleviates neuropathic pain in chronic constriction injury rat models via the microRNA‐140‐3p/Jagged‐1 peptide/Notch signaling pathway
Cheng Fang, Wei Qin, Aixing Yang, et al.
Synapse (2021) Vol. 75, Iss. 10
Closed Access | Times Cited: 12
Research progress on miR-124-3p in the field of kidney disease
Guanting Chen, Yaoxian Wang, Linqi Zhang, et al.
BMC Nephrology (2024) Vol. 25, Iss. 1
Open Access | Times Cited: 1
Differential Expression of Long Non-Coding RNAs and Their Role in Rodent Neuropathic Pain Models
Songchao Xu, He Dong, Yang Zhao, et al.
Journal of Pain Research (2021) Vol. Volume 14, pp. 3935-3950
Open Access | Times Cited: 8
miR-124-3p Combined with ANGPTL2 Has High Diagnostic Values for Obese and Nonobese Polycystic Ovary Syndrome
Hongmei Dai, Fangting Liu, Jianshu Lu, et al.
International Journal of Endocrinology (2022) Vol. 2022, pp. 1-11
Open Access | Times Cited: 5
LncRNA FTX ameliorates neuropathic pain by targeting miR-320a in a rat model of chronic constriction injury
Zhisheng Lu, Yijue Zhang, Yunze Li
Folia Neuropathologica (2023) Vol. 61, Iss. 3, pp. 291-300
Open Access | Times Cited: 2
JAG‐1/Notch signaling axis in the spinal cord contributes to bone cancer pain in rats
Jie Fu, Baoxia Zhao, Ge Luo, et al.
Journal of Neurochemistry (2023) Vol. 166, Iss. 4, pp. 747-762
Closed Access | Times Cited: 2
Long non-coding RNA Snhg16 Lessens Ozone Curative Effect on Chronic Constriction Injury mice via microRNA-719/SCN1A axis
Jianning Yue, Qi Wang, Wenxing Zhao, et al.
Molecular Biotechnology (2023) Vol. 66, Iss. 9, pp. 2273-2286
Closed Access | Times Cited: 1
JAG-1/Notch signaling axis contributes to the maintenance of bone cancer pain in female rats via spinal astrocyte-neuron crosstalk
杰 伏, Baoxia Zhao, Huadong Ni, et al.
Research Square (Research Square) (2022)
Open Access
Propofol Alleviates Neuropathic Pain in CCI Rat Model via the miR-140-3p/JAG1/Notch Signaling Pathway
Cheng Fang, Wei Qin, Aixing Yang, et al.
Research Square (Research Square) (2021)
Open Access
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