OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Inhibition of focal adhesion kinase/paxillin axis by caffeic acid phenethyl ester restrains aggressive behaviors of head and neck squamous cell carcinoma in vitro
Hyun‐Ju Yu, Ji‐Ae Shin, Sung‐Dae Cho
Archives of Oral Biology (2022) Vol. 146, pp. 105611-105611
Closed Access | Times Cited: 6

Showing 6 citing articles:

Dietary Polyphenols Effects on Focal Adhesion Plaques and Metalloproteinases in Cancer Invasiveness
Raffaele Carrano, Martina Grande, Eleonora Leti Maggio, et al.
Biomedicines (2024) Vol. 12, Iss. 3, pp. 482-482
Open Access | Times Cited: 4

Plant phenolics inhibit focal adhesion kinase and suppress host cell invasion by uropathogenic Escherichia coli
Adam Lewis, Amanda C. Richards, Alejandra A. Mendez, et al.
Infection and Immunity (2024) Vol. 92, Iss. 5
Closed Access | Times Cited: 3

Signal Transduction Mechanisms of Focal Adhesions: Src and FAK-Mediated Cell Response
Kazuo Katoh
Frontiers in Bioscience-Landmark (2024) Vol. 29, Iss. 11
Open Access

In Silico and In Vitro Anticancer Effects of Caffeic Acid Phenethyl Ester on Pancreatic Adenocarcinoma Cells
Zübeyde Tanrıverdi, Burak Kuzu, Ergül Eyol, et al.
Fabad Journal of Pharmaceutical Sciences (2023)
Open Access

Plant Phenolics Inhibit Focal Adhesion Kinase and Suppress Host Cell Invasion by UropathogenicEscherichia coli
Adam Lewis, Amanda C. Richards, Alejandra A. Mendez, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2023)
Open Access

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