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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Endosomal-lysosomal dysfunction in metabolic diseases and Alzheimer's disease
Michael F. Almeida, Ben A. Bahr, Stephen T. Kinsey
International review of neurobiology (2020), pp. 303-324
Open Access | Times Cited: 24

Showing 24 citing articles:

Mechanisms of autophagy–lysosome dysfunction in neurodegenerative diseases
Ralph A. Nixon, David C. Rubinsztein
Nature Reviews Molecular Cell Biology (2024) Vol. 25, Iss. 11, pp. 926-946
Closed Access | Times Cited: 50
Lysosomal dysfunction in Down syndrome and Alzheimer mouse models is caused by v-ATPase inhibition by Tyr 682 -phosphorylated APP βCTF
Eunju Im, Ying Jiang, Philip Stavrides, et al.
Science Advances (2023) Vol. 9, Iss. 30
Open Access | Times Cited: 38
Neprilysin inhibitors and risk of Alzheimer's disease: A future perspective
Naif H. Ali, Hayder M. Al‐kuraishy, Ali I. Al‐Gareeb, et al.
Journal of Cellular and Molecular Medicine (2023) Vol. 28, Iss. 2
Open Access | Times Cited: 23
Endo-lysosomal dysfunction in neurodegenerative diseases: opinion on current progress and future direction in the use of exosomes as biomarkers
Mathieu Herman, Grace W. Randall, Julia L. Spiegel, et al.
Philosophical Transactions of the Royal Society B Biological Sciences (2024) Vol. 379, Iss. 1899
Open Access | Times Cited: 10
Alzheimer's Disease Biomarker Analysis Using Targeted Mass Spectrometry
Johan Gobom, Ann Brinkmalm, Gunnar Brinkmalm, et al.
Molecular & Cellular Proteomics (2024) Vol. 23, Iss. 2, pp. 100721-100721
Open Access | Times Cited: 9
The Biology of Lysosomes: From Order to Disorder
Olga Amaral, Mariana Martins, Ana Rita Oliveira, et al.
Biomedicines (2023) Vol. 11, Iss. 1, pp. 213-213
Open Access | Times Cited: 20
The Role of Cysteine Protease Cathepsins B, H, C, and X/Z in Neurodegenerative Diseases and Cancer
Veronika Stoka, Olga Vasiljeva, Hiroshi Nakanishi, et al.
International Journal of Molecular Sciences (2023) Vol. 24, Iss. 21, pp. 15613-15613
Open Access | Times Cited: 16
From Metabolic Syndrome to Neurological Diseases: Role of Autophagy
Jessica Maiuolo, Micaela Gliozzi, Vincenzo Musolino, et al.
Frontiers in Cell and Developmental Biology (2021) Vol. 9
Open Access | Times Cited: 32
Lysosomes’ fallback strategies: more than just survival or death
Quan Wang, Ruolin Wang, Hanqing Hu, et al.
Frontiers in Cell and Developmental Biology (2025) Vol. 13
Open Access
Preclinical neuropharmacological effects of Centella asiatica-derived asiaticoside and madecassoside in Alzheimer's disease
Narudol Teerapattarakan, Utcharaporn Kamsrijai, Adchara Janyou, et al.
South African Journal of Botany (2025) Vol. 180, pp. 171-184
Closed Access
Excessive Alcohol Use as a Risk Factor for Alzheimer’s Disease: Epidemiological and Preclinical Evidence
Paige Anton, Nicole Maphis, David N. Linsenbardt, et al.
Advances in experimental medicine and biology (2025), pp. 211-242
Closed Access
The probable role of tissue plasminogen activator/neuroserpin axis in Alzheimer’s disease: a new perspective
Naif H. Ali, Hayder M. Al‐kuraishy, Ali I. Al‐Gareeb, et al.
Acta Neurologica Belgica (2023) Vol. 124, Iss. 2, pp. 377-388
Open Access | Times Cited: 10
Glucose starvation causes ferroptosis-mediated lysosomal dysfunction
K. Miki, Mikako Yagi, Dongchon Kang, et al.
iScience (2024) Vol. 27, Iss. 5, pp. 109735-109735
Open Access | Times Cited: 3
Cathepsins and Parkinson’s disease: insights from Mendelian randomization analyses
Aishanjiang Yusufujiang, Shan Zeng, Hongyan Li
Frontiers in Aging Neuroscience (2024) Vol. 16
Open Access | Times Cited: 3
c-Abl kinase at the crossroads of healthy synaptic remodeling and synaptic dysfunction in neurodegenerative diseases
Alejandra Álvarez, DanielaA Gutiérrez, América Chandía-Cristi, et al.
Neural Regeneration Research (2022) Vol. 18, Iss. 2, pp. 237-237
Open Access | Times Cited: 15
Broadening Horizons: Exploring the Cathepsin Family as Therapeutic Targets for Alzheimer's Disease
Xiaohui Liu, Xiaotong Liu, Yue Wu, et al.
Aging and Disease (2024)
Open Access | Times Cited: 2
Loss of neuronal lysosomal acid lipase drives amyloid pathology in Alzheimer’s disease
Alexandra Barnett, Lamar Dawkins, Jian Zou, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access | Times Cited: 2
Tangzhiqing decoction attenuates cognitive dysfunction of mice with type 2 diabetes by regulating AMPK/mTOR autophagy signaling pathway
Wenqiang Yao, Qing Zhang, Yun Zhao, et al.
Journal of Ethnopharmacology (2023) Vol. 321, pp. 117536-117536
Closed Access | Times Cited: 4
In situ stoichiometry amounts of p62 and poly-ubiquitin exceed the increase of alpha-synuclein during degeneration of catecholamine cells induced by autophagy inhibition in vitro
Paola Lenzi, Gloria Lazzeri, Michela Ferrucci, et al.
Journal of Neural Transmission (2024)
Open Access | Times Cited: 1
Lysosomal dysfunction in Down Syndrome and Alzheimer mouse models is caused by selective v-ATPase inhibition by Tyr682phosphorylated APP βCTF
Eunju Im, Ying Jiang, Philip Stavrides, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2022)
Open Access | Times Cited: 6
Lysosomes: multifunctional compartments ruled by a complex regulatory network
Jonathan Martínez‐Fábregas, Joaquin Tamargo‐Azpilicueta, Irene Díaz‐Moreno
FEBS Open Bio (2022) Vol. 12, Iss. 4, pp. 758-774
Open Access | Times Cited: 4
A kaleidoscopic view of extracellular vesicles in lysosomal storage disorders
Charlotte V Hegeman, Olivier G. de Jong, Magdalena J. Lorenowicz
Extracellular Vesicles and Circulating Nucleic Acids (2022) Vol. 3, Iss. 4
Open Access | Times Cited: 4
Inhibition of CysLTR1 reduces the levels of aggregated proteins in retinal pigment epithelial cells
Andreas Koller, Susanne M. Brunner, Julia Preishuber‐Pflügl, et al.
Scientific Reports (2023) Vol. 13, Iss. 1
Open Access | Times Cited: 1
Lysosomal Dysfunction in Down Syndrome and Alzheimer Mouse Models is Caused by Selective V-Atpase Inhibition by Tyr <sup>682</sup> Phosphorylated APP βCTF
Eunju Im, Ying Jiang, Philip Stavrides, et al.
SSRN Electronic Journal (2022)
Closed Access
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