OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

The Multifaceted Contributions of Chromatin to HIV-1 Integration, Transcription, and Latency
Elisa De Crignis, Tokameh Mahmoudi
International review of cell and molecular biology (2016), pp. 197-252
Closed Access | Times Cited: 18

Showing 18 citing articles:

Distinct chromatin functional states correlate with HIV latency reactivation in infected primary CD4+ T cells
Emilie Battivelli, Matthew S. Dahabieh, Mohamed Abdel‐Mohsen, et al.
eLife (2018) Vol. 7
Open Access | Times Cited: 166

Transcription: Insights From the HIV-1 Promoter
Enrico Ne, Robert‐Jan Palstra, Tokameh Mahmoudi
International review of cell and molecular biology (2017), pp. 191-243
Closed Access | Times Cited: 65

Small Molecule Targeting of Specific BAF (mSWI/SNF) Complexes for HIV Latency Reversal
Christine Marian, Mateusz Stoszko, Lili Wang, et al.
Cell chemical biology (2018) Vol. 25, Iss. 12, pp. 1443-1455.e14
Open Access | Times Cited: 48

A broad drug arsenal to attack a strenuous latent HIV reservoir
Mateusz Stoszko, Enrico Ne, Erik Abner, et al.
Current Opinion in Virology (2019) Vol. 38, pp. 37-53
Open Access | Times Cited: 34

A Review of Current Strategies Towards the Elimination of Latent HIV-1 and Subsequent HIV-1 Cure
Edward K. Maina, Asma Adan, Haddison Mureithi, et al.
Current HIV Research (2020) Vol. 19, Iss. 1, pp. 14-26
Open Access | Times Cited: 30

Episomal HBV persistence within transcribed host nuclear chromatin compartments involves HBx
Kai O. Hensel, Franziska Cantner, Felix Bangert, et al.
Epigenetics & Chromatin (2018) Vol. 11, Iss. 1
Open Access | Times Cited: 30

HibeRNAtion: HIV-1 RNA Metabolism and Viral Latency
Raquel Crespo, Shringar Rao, Tokameh Mahmoudi
Frontiers in Cellular and Infection Microbiology (2022) Vol. 12
Open Access | Times Cited: 13

HIV-1 Proviral Genome Engineering with CRISPR-Cas9 for Mechanistic Studies
Usman Hyder, Ashutosh Shukla, Ashwini Challa, et al.
Viruses (2024) Vol. 16, Iss. 2, pp. 287-287
Open Access | Times Cited: 2

HIV-Tocky system to visualize proviral expression dynamics
Omnia Reda, Kazuaki Monde, Kenji Sugata, et al.
Communications Biology (2024) Vol. 7, Iss. 1
Open Access | Times Cited: 2

Anti-DFS70 autoantibodies in HIV-1-positive individuals
María Dolores Pérez, Danielle Azoulay
Current Opinion in Rheumatology (2018) Vol. 30, Iss. 4, pp. 361-364
Closed Access | Times Cited: 8

Selective elimination of host cells harboring replication-competent human immunodeficiency virus reservoirs: a promising therapeutic strategy for HIV cure
Silvère D. Zaongo, Yue Wang, Ping Ma, et al.
Chinese Medical Journal (2021) Vol. 134, Iss. 23, pp. 2776-2787
Open Access | Times Cited: 7

Chromatin Functional States Correlate with HIV Latency Reversal in Infected Primary CD4+T cells
Emilie Battivelli, Matthew S. Dahabieh, Mohamed Abdel‐Mohsen, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2018)
Open Access | Times Cited: 6

Gliotoxin, identified from a screen of fungal metabolites, disrupts 7SK snRNP, releases P-TEFb and reverses HIV-1 latency
Mateusz Stoszko, Abdullah M. S. Al‐Hatmi, Anton Škríba, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2019)
Open Access | Times Cited: 2

HIV-Tocky system to visualize proviral expression dynamics
Omnia Reda, Kazuaki Monde, Kenji Sugata, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2023)
Open Access

HIV-Tocky system to visualize proviral expression dynamics
Yorifumi Satou, Omnia Reda, Kazuaki Monde, et al.
Research Square (Research Square) (2023)
Open Access

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