OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Resolution-promoting autacoids demonstrate promising cardioprotective effects against heart diseases
Roddy Hiram
Molecular Biology Reports (2022) Vol. 49, Iss. 6, pp. 5179-5197
Open Access | Times Cited: 15

Showing 15 citing articles:

Inflammatory signalling in atrial cardiomyocytes: a novel unifying principle in atrial fibrillation pathophysiology
Dobromir Dobrev, Jordi Heijman, Roddy Hiram, et al.
Nature Reviews Cardiology (2022) Vol. 20, Iss. 3, pp. 145-167
Open Access | Times Cited: 151

Specialized pro-resolving mediators in vascular inflammation and atherosclerotic cardiovascular disease
Gabrielle Fredman, Charles N. Serhan
Nature Reviews Cardiology (2024) Vol. 21, Iss. 11, pp. 808-823
Closed Access | Times Cited: 41

An inflammation resolution–promoting intervention prevents atrial fibrillation caused by left ventricular dysfunction
Roddy Hiram, Feng Xiong, Patrice Naud, et al.
Cardiovascular Research (2023) Vol. 120, Iss. 4, pp. 345-359
Closed Access | Times Cited: 12

Atrial cardiomyocytes contribute to the inflammatory status associated with atrial fibrillation in right heart disease
Ewen Le Quilliec, Charles-Alexandre LeBlanc, Orlane Neuilly, et al.
EP Europace (2024) Vol. 26, Iss. 4
Open Access | Times Cited: 3

Maresin1 Ameliorates Sepsis-Induced Microglial Neuritis Induced through Blocking TLR4-NF-κ B-NLRP3 Signaling Pathway
Huiping Wu, Ying Wang, Haiyan Fu, et al.
Journal of Personalized Medicine (2023) Vol. 13, Iss. 3, pp. 534-534
Open Access | Times Cited: 8

Colchicine for the prevention of atrial fibrillation: Why do some studies say ‘yes’, and others say ‘no’?
Abir Attia, Roddy Hiram
International Journal of Cardiology (2024) Vol. 408, pp. 132110-132110
Closed Access | Times Cited: 2

Pollutants, including Organophosphorus and Organochloride Pesticides, May Increase the Risk of Cardiac Remodeling and Atrial Fibrillation: A Narrative Review
Ewen Le Quilliec, Alexia Fundere, Doa’a Al-u’datt, et al.
Biomedicines (2023) Vol. 11, Iss. 9, pp. 2427-2427
Open Access | Times Cited: 6

Evidence of Failed Resolution Mechanisms in Arrhythmogenic Inflammation, Fibrosis and Right Heart Disease
Rim Younes, Charles-Alexandre LeBlanc, Roddy Hiram
Biomolecules (2022) Vol. 12, Iss. 5, pp. 720-720
Open Access | Times Cited: 9

Comprehensive Role of GDF15 in Inhibiting Adipogenesis and Hyperlipidemia, Enhancing Cardiovascular Health and Alleviating Inflammation in Metabolic Disorders
Nikita Nayak, Tuhin Mukherjee, Ashok Kumar Pattnaik
Current Pharmaceutical Design (2024) Vol. 30, Iss. 30, pp. 2387-2399
Closed Access | Times Cited: 1

Daily Exposure to Chlordecone, an Organochlorine Pesticide, Increases Cardiac Fibrosis and Atrial Fibrillation Vulnerability
Alexia Fundere, Andrew Rose, Feng Xiong, et al.
Journal of Hazardous Materials (2024) Vol. 478, pp. 135533-135533
Open Access | Times Cited: 1

The cross-talk between the cGAS-STING signaling pathway and chronic inflammation in the development of musculoskeletal disorders
Alexander Kalinkovich, Gregory Livshits
Ageing Research Reviews (2024), pp. 102602-102602
Closed Access | Times Cited: 1

Ultrasensitive dose-response for asbestos cancer risk implied by new inflammation-mutation model
Kenneth T. Bogen
Environmental Research (2023) Vol. 230, pp. 115047-115047
Open Access | Times Cited: 2

The duration of atrial fibrillation might be associated with right heart disease severity
Roddy Hiram
International Journal of Cardiology (2023) Vol. 400, pp. 131689-131689
Closed Access | Times Cited: 2

Exercise training attenuates cardiac dysfunction induced by excessive sympathetic activation through an AMPK-KLF4-FMO2 axis
Shiyu Fan, Mingming Zhao, Kang Wang, et al.
Journal of Molecular and Cellular Cardiology (2024) Vol. 197, pp. 136-149
Closed Access

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