OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

De novo CACAN1D Ca2+ channelopathies: clinical phenotypes and molecular mechanism
Nadine J. Ortner, Teresa Kaserer, J. Nathan Copeland, et al.
Pflügers Archiv - European Journal of Physiology (2020) Vol. 472, Iss. 7, pp. 755-773
Open Access | Times Cited: 53

Showing 1-25 of 53 citing articles:

Genetics of Primary Aldosteronism
Ute I. Scholl
Hypertension (2022) Vol. 79, Iss. 5, pp. 887-897
Open Access | Times Cited: 64

Primary aldosteronism: molecular medicine meets public health
Elena Azizan, William Drake, Morris J. Brown
Nature Reviews Nephrology (2023) Vol. 19, Iss. 12, pp. 788-806
Open Access | Times Cited: 23

A Review of the CACNA Gene Family: Its Role in Neurological Disorders
Oliwia Szymanowicz, Artur Drużdż, Bartosz Słowikowski, et al.
Diseases (2024) Vol. 12, Iss. 5, pp. 90-90
Open Access | Times Cited: 11

Familial hyperaldosteronism: an European Reference Network on Rare Endocrine Conditions clinical practice guideline
Paolo Mulatero, Ute I. Scholl, Carlos Fardella, et al.
European Journal of Endocrinology (2024) Vol. 190, Iss. 4, pp. G1-G14
Open Access | Times Cited: 7

CACNA1I gain-of-function mutations differentially affect channel gating and cause neurodevelopmental disorders
Yousra El Ghaleb, Pauline E. Schneeberger, Monica L. Fernández‐Quintero, et al.
Brain (2021) Vol. 144, Iss. 7, pp. 2092-2106
Open Access | Times Cited: 38

Role of Ca2+ in healthy and pathologic cardiac function: from normal excitation–contraction coupling to mutations that cause inherited arrhythmia
Joshua A. Keefe, Oliver M. Moore, Kevin S. Ho, et al.
Archives of Toxicology (2022) Vol. 97, Iss. 1, pp. 73-92
Open Access | Times Cited: 23

Treating Primary Aldosteronism-Induced Hypertension: Novel Approaches and Future Outlooks
Nathan Mullen, James Curneen, Padraig Donlon, et al.
Endocrine Reviews (2023) Vol. 45, Iss. 1, pp. 125-170
Open Access | Times Cited: 13

Inactivation of CaV1 and CaV2 channels
Worawan B. Limpitikul, Ivy E. Dick
The Journal of General Physiology (2025) Vol. 157, Iss. 2
Closed Access

Small Molecules as Modulators of Voltage-Gated Calcium Channels in Neurological Disorders: State of the Art and Perspectives
Stefano Lanzetti, Valentina Di Biase
Molecules (2022) Vol. 27, Iss. 4, pp. 1312-1312
Open Access | Times Cited: 21

The human channel gating–modifying A749G CACNA1D (Cav1.3) variant induces a neurodevelopmental syndrome–like phenotype in mice
Nadine J. Ortner, Anupam Sah, Enrica Paradiso, et al.
JCI Insight (2023) Vol. 8, Iss. 20
Open Access | Times Cited: 12

Cav1.3‐selective inhibitors of voltage‐gated L‐type Ca2+ channels: Fact or (still) fiction?
Ludovica Filippini, Nadine J. Ortner, Teresa Kaserer, et al.
British Journal of Pharmacology (2023) Vol. 180, Iss. 10, pp. 1289-1303
Open Access | Times Cited: 11

Evaluation of Aldosterone Suppression by Cinnarizine, a Putative Cav1.3 Inhibitor
Elisabeth Ng, Yun-Ni Lee, Angela E. Taylor, et al.
The Journal of Clinical Endocrinology & Metabolism (2025)
Open Access

Genetic Alterations in Atypical Cerebral Palsy Identified Through Chromosomal Microarray and Exome Sequencing
Ji Yoon Han, Jin Gwack, Jong Hun Kim, et al.
International Journal of Molecular Sciences (2025) Vol. 26, Iss. 7, pp. 2929-2929
Open Access

CACNA1D-Related Channelopathies: From Hypertension to Autism
Nadine J. Ortner
Handbook of experimental pharmacology (2023), pp. 183-225
Closed Access | Times Cited: 10

A Novel De Novo Gain-of-Function CACNA1D Variant in Neurodevelopmental Disease With Congenital Tremor, Seizures, and Hypotonia
Fabian Dannenberg, Arpad von Moers, Petra Bittigau, et al.
Neurology Genetics (2024) Vol. 10, Iss. 5
Open Access | Times Cited: 3

Isradipine therapy in Cacna1dIle772Met/+ mice ameliorates primary aldosteronism and neurologic abnormalities
Gabriel Stölting, Hoang An Dinh, Marina Volkert, et al.
JCI Insight (2023) Vol. 8, Iss. 20
Open Access | Times Cited: 7

Pathogenicity of de novo CACNA1D Ca2+ channel variants predicted from sequence co-variation
Xuechen Tang, Nadine J. Ortner, Yuliia V. Nikonishyna, et al.
European Journal of Human Genetics (2024) Vol. 32, Iss. 9, pp. 1065-1073
Open Access | Times Cited: 2

The L‐type calcium channel CaV1.3: A potential target for cancer therapy
Xuerun Liu, Boqiang Shen, Jingyi Zhou, et al.
Journal of Cellular and Molecular Medicine (2024) Vol. 28, Iss. 19
Open Access | Times Cited: 2

Germline de novo variant F747S extends the phenotypic spectrum ofCACNA1DCa2+ channelopathies
Ferenc Török, Kamer Tezcan, Ludovica Filippini, et al.
Human Molecular Genetics (2022) Vol. 32, Iss. 5, pp. 847-859
Open Access | Times Cited: 12

CACNA1D Gene Polymorphisms Associate With Increased Blood Pressure and Salt Sensitivity of Blood Pressure in White Individuals
Ana Maria Stanton, Mahyar Heydarpour, Jonathan S. Williams, et al.
Hypertension (2023) Vol. 80, Iss. 12, pp. 2665-2673
Open Access | Times Cited: 6

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