OpenAlex Citation Counts

OpenAlex Citations Logo

OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Mitochondrial bioenergetic deficits in C9orf72 amyotrophic lateral sclerosis motor neurons cause dysfunctional axonal homeostasis
Arpan R. Mehta, Jenna M. Gregory, Owen Dando, et al.
Acta Neuropathologica (2021) Vol. 141, Iss. 2, pp. 257-279
Open Access | Times Cited: 103

Showing 1-25 of 103 citing articles:

Functional roles of reactive astrocytes in neuroinflammation and neurodegeneration
Rickie Patani, Giles E. Hardingham, Shane A. Liddelow
Nature Reviews Neurology (2023) Vol. 19, Iss. 7, pp. 395-409
Closed Access | Times Cited: 233

Mitochondria, energy, and metabolism in neuronal health and disease
Diogo Trigo, Catarina Avelar, Miguel X. Fernandes, et al.
FEBS Letters (2022) Vol. 596, Iss. 9, pp. 1095-1110
Closed Access | Times Cited: 108

Genome-wide identification of the genetic basis of amyotrophic lateral sclerosis
Sai Zhang, Johnathan Cooper‐Knock, Annika K. Weimer, et al.
Neuron (2022) Vol. 110, Iss. 6, pp. 992-1008.e11
Open Access | Times Cited: 96

Genetics of amyotrophic lateral sclerosis: seeking therapeutic targets in the era of gene therapy
Naoki Suzuki, Ayumi Nishiyama, Hitoshi Warita, et al.
Journal of Human Genetics (2022) Vol. 68, Iss. 3, pp. 131-152
Open Access | Times Cited: 92

Mitochondrial Dysfunction in Neurodegenerative Diseases: Mechanisms and Corresponding Therapeutic Strategies
Kai Meng, Hongyun Jia, Xiaoqing Hou, et al.
Biomedicines (2025) Vol. 13, Iss. 2, pp. 327-327
Open Access | Times Cited: 2

C9orf72-derived arginine-containing dipeptide repeats associate with axonal transport machinery and impede microtubule-based motility
Laura Fumagalli, Florence L. Young, Steven Boeynaems, et al.
Science Advances (2021) Vol. 7, Iss. 15
Open Access | Times Cited: 84

The Link between Oxidative Stress, Redox Status, Bioenergetics and Mitochondria in the Pathophysiology of ALS
Elena Obrador, Rosario Salvador‐Palmer, Rafael López‐Blanch, et al.
International Journal of Molecular Sciences (2021) Vol. 22, Iss. 12, pp. 6352-6352
Open Access | Times Cited: 66

Human stem cell models of neurodegeneration: From basic science of amyotrophic lateral sclerosis to clinical translation
Elisa Giacomelli, Björn F. Vahsen, Elizabeth L. Calder, et al.
Cell stem cell (2022) Vol. 29, Iss. 1, pp. 11-35
Open Access | Times Cited: 66

The Role of Mitochondrial Dysfunction and ER Stress in TDP-43 and C9ORF72 ALS
Ruxandra Dafinca, Paola Barbagallo, Kevin Talbot
Frontiers in Cellular Neuroscience (2021) Vol. 15
Open Access | Times Cited: 59

The use of fibroblasts as a valuable strategy for studying mitochondrial impairment in neurological disorders
Margrethe A. Olesen, Francisca Villavicencio-Tejo, Rodrigo A. Quintanilla
Translational Neurodegeneration (2022) Vol. 11, Iss. 1
Open Access | Times Cited: 38

Nutritional and metabolic factors in amyotrophic lateral sclerosis
Albert C. Ludolph, Luc Dupuis, Edward J. Kasarskis, et al.
Nature Reviews Neurology (2023) Vol. 19, Iss. 9, pp. 511-524
Closed Access | Times Cited: 29

Blood-CNS barrier dysfunction in amyotrophic lateral sclerosis: Proposed mechanisms and clinical implications
Moritz Steinruecke, Rebecca Murphy Lonergan, Bhuvaneish T. Selvaraj, et al.
Journal of Cerebral Blood Flow & Metabolism (2023) Vol. 43, Iss. 5, pp. 642-654
Open Access | Times Cited: 27

Cell-autonomous immune dysfunction driven by disrupted autophagy in C9orf72 -ALS iPSC-derived microglia contributes to neurodegeneration
Poulomi Banerjee, Arpan R. Mehta, Raja Sekhar Nirujogi, et al.
Science Advances (2023) Vol. 9, Iss. 16
Open Access | Times Cited: 27

The Role of PGC-1α-Mediated Mitochondrial Biogenesis in Neurons
Mengjie Chen, Ru-Yu Yan, Jian-Sheng Luo, et al.
Neurochemical Research (2023) Vol. 48, Iss. 9, pp. 2595-2606
Closed Access | Times Cited: 26

Molecular hallmarks of ageing in amyotrophic lateral sclerosis
Cyril J. Jagaraj, Sina Shadfar, Sara Assar Kashani, et al.
Cellular and Molecular Life Sciences (2024) Vol. 81, Iss. 1
Open Access | Times Cited: 11

Study insights in the role of PGC-1α in neurological diseases: mechanisms and therapeutic potential
Mibo Tang, Yixuan Liu, Zhengwei Hu, et al.
Frontiers in Aging Neuroscience (2025) Vol. 16
Open Access | Times Cited: 1

Intracellular energy controls dynamics of stress-induced ribonucleoprotein granules
Tao Wang, Xibin Tian, Han Byeol Kim, et al.
Nature Communications (2022) Vol. 13, Iss. 1
Open Access | Times Cited: 34

Disturb mitochondrial associated proteostasis: Neurodegeneration and imperfect ageing
Yuvraj Anandrao Jagtap, Prashant Kumar, Sumit Kinger, et al.
Frontiers in Cell and Developmental Biology (2023) Vol. 11
Open Access | Times Cited: 19

Evidence of Metabolic Dysfunction in Amyotrophic Lateral Sclerosis (ALS) Patients and Animal Models
Katarina Maksimovic, Mohieldin Youssef, Justin You, et al.
Biomolecules (2023) Vol. 13, Iss. 5, pp. 863-863
Open Access | Times Cited: 19

Loss of function of the ALS-associated NEK1 kinase disrupts microtubule homeostasis and nuclear import
Jacob R. Mann, Elizabeth D. McKenna, Darilang Mawrie, et al.
Science Advances (2023) Vol. 9, Iss. 33
Open Access | Times Cited: 19

Mitochondria, a Key Target in Amyotrophic Lateral Sclerosis Pathogenesis
Emmanuelle C. Genin, Mélanie Abou-Ali, Véronique Paquis‐Flucklinger
Genes (2023) Vol. 14, Iss. 11, pp. 1981-1981
Open Access | Times Cited: 19

TDP-43 Proteinopathy Specific Biomarker Development
Isabell Cordts, Annika Wachinger, Carlo Scialò, et al.
Cells (2023) Vol. 12, Iss. 4, pp. 597-597
Open Access | Times Cited: 17

Synaptic disruption and CREB‐regulated transcription are restored by K + channel blockers in ALS
Alberto Catanese, Sandeep Rajkumar, Doron D. Sommer, et al.
EMBO Molecular Medicine (2021) Vol. 13, Iss. 7
Open Access | Times Cited: 34

PolyGA targets the ER stress-adaptive response by impairing GRP75 function at the MAM in C9ORF72-ALS/FTD
Federica Pilotto, Alexander Schmitz, Niran Maharjan, et al.
Acta Neuropathologica (2022) Vol. 144, Iss. 5, pp. 939-966
Open Access | Times Cited: 23

Page 1 - Next Page

Scroll to top