Open Access Helper

OpenAlex Citation Counts

OpenAlex Citations Logo

OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Cardiac fibrosis
Nikolaos G. Frangogiannis
Cardiovascular Research (2020) Vol. 117, Iss. 6, pp. 1450-1488
Open Access | Times Cited: 717

Showing 76-100 of 717 citing articles:

Comparative phenotyping of C57BL/6J substrains reveals distinctive patterns of cardiac aging
Sophia Walter, Peter Baumgarten, Niklas Hegemann, et al.
GeroScience (2025)
Open Access | Times Cited: 1
Mosaic loss of Y chromosome and mortality after coronary angiography
Michael Weyrich, Stephen Zewinger, Tamim Sarakpi, et al.
European Heart Journal (2025)
Open Access | Times Cited: 1
A heart-brain-spleen axis controls cardiac remodeling to hypertensive stress
Sara Perrotta, Lorenzo Carnevale, Marialuisa Perrotta, et al.
Immunity (2025)
Open Access | Times Cited: 1
Tissue macrophages: origin, heterogenity, biological functions, diseases and therapeutic targets
Fan Guan, Ruixuan Wang, Zhenjie Yi, et al.
Signal Transduction and Targeted Therapy (2025) Vol. 10, Iss. 1
Open Access | Times Cited: 1
SLC31A1 loss depletes mitochondrial copper and promotes cardiac fibrosis
Bin Tu, Kai Song, Ze‐Yu Zhou, et al.
European Heart Journal (2025)
Closed Access | Times Cited: 1
PGRMC2 is a pressure-volume regulator critical for myocardial responses to stress in mice
Farideh Amirrad, Vivian La, Sharareh Ohadi, et al.
Nature Communications (2025) Vol. 16, Iss. 1
Open Access | Times Cited: 1
Electrophysiological Consequences of Cardiac Fibrosis
Sander Verheule, Ulrich Schotten
Cells (2021) Vol. 10, Iss. 11, pp. 3220-3220
Open Access | Times Cited: 54
Fibroblast‐mediated intercellular crosstalk in the healthy and diseased heart
Luka Nicin, Julian U. G. Wagner, Guillermo Luxán, et al.
FEBS Letters (2021) Vol. 596, Iss. 5, pp. 638-654
Open Access | Times Cited: 47
circCELF1 Inhibits Myocardial Fibrosis by Regulating the Expression of DKK2 Through FTO/m6A and miR-636
Xuexun Li, Bin Mu, Xi Li, et al.
Journal of Cardiovascular Translational Research (2022) Vol. 15, Iss. 5, pp. 998-1009
Closed Access | Times Cited: 36
Early Protective Role of Inflammation in Cardiac Remodeling and Heart Failure: Focus on TNFα and Resident Macrophages
Sophie Besse, Sophie Nadaud, Elise Balse, et al.
Cells (2022) Vol. 11, Iss. 7, pp. 1249-1249
Open Access | Times Cited: 35
Treatment of cardiac fibrosis: from neuro-hormonal inhibitors to CAR-T cell therapy
Paolo Morfino, Alberto Aimo, Vincenzo Castiglione, et al.
Heart Failure Reviews (2022)
Open Access | Times Cited: 35
CXCR4-dependent macrophage-to-fibroblast signaling contributes to cardiac diastolic dysfunction in heart failure with preserved ejection fraction
Ning Zhang, Qunchao Ma, Yayu You, et al.
International Journal of Biological Sciences (2022) Vol. 18, Iss. 3, pp. 1271-1287
Open Access | Times Cited: 34
Integrins in cardiac fibrosis
Ruoshui Li, Nikolaos G. Frangogiannis
Journal of Molecular and Cellular Cardiology (2022) Vol. 172, pp. 1-13
Open Access | Times Cited: 34
Novel Therapies for the Treatment of Cardiac Fibrosis Following Myocardial Infarction
Kamila Raziyeva, Yevgeniy Kim, Zharylkasyn Zharkinbekov, et al.
Biomedicines (2022) Vol. 10, Iss. 9, pp. 2178-2178
Open Access | Times Cited: 33
CCL17 acts as a novel therapeutic target in pathological cardiac hypertrophy and heart failure
Yang Zhang, Yicong Ye, Xiaoqiang Tang, et al.
The Journal of Experimental Medicine (2022) Vol. 219, Iss. 8
Open Access | Times Cited: 30
The Atrium in Atrial Fibrillation – A Clinical Review on How to Manage Atrial Fibrotic Substrates
Pedro Silva Cunha, Sérgio Laranjo, Jordi Heijman, et al.
Frontiers in Cardiovascular Medicine (2022) Vol. 9
Open Access | Times Cited: 30
Global Research Trends on Ventricular Remodeling: A Bibliometric Analysis From 2012 to 2022
Tiantian Meng, Yanchun Wang, Jingyi Ding, et al.
Current Problems in Cardiology (2022) Vol. 47, Iss. 11, pp. 101332-101332
Open Access | Times Cited: 29
Exosomes and Exosomal Cargos: A Promising World for Ventricular Remodeling Following Myocardial Infarction
Jiacheng Fang, Yuxuan Zhang, Delong Chen, et al.
International Journal of Nanomedicine (2022) Vol. Volume 17, pp. 4699-4719
Open Access | Times Cited: 29
Fucoxanthin ameliorated myocardial fibrosis in STZ-induced diabetic rats and cell hypertrophy in HG-induced H9c2 cells by alleviating oxidative stress and restoring mitophagy
Dongxiao Zheng, Linlin Chen, Guoping Li, et al.
Food & Function (2022) Vol. 13, Iss. 18, pp. 9559-9575
Closed Access | Times Cited: 28
METTL3 boosts glycolysis and cardiac fibroblast proliferation by increasing AR methylation
Yang Zhou, Kai Song, Bin Tu, et al.
International Journal of Biological Macromolecules (2022) Vol. 223, pp. 899-915
Closed Access | Times Cited: 28
Cardiomyocyte NOX4 regulates resident macrophage-mediated inflammation and diastolic dysfunction in stress cardiomyopathy
Aleksandr E. Vendrov, Xiao Han, Andrey Lozhkin, et al.
Redox Biology (2023) Vol. 67, pp. 102937-102937
Open Access | Times Cited: 22
Senescent cardiac fibroblasts: A key role in cardiac fibrosis
José Miguel Osorio, Claudio Espinoza-Pérez, Constanza Rimassa-Taré, et al.
Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease (2023) Vol. 1869, Iss. 4, pp. 166642-166642
Open Access | Times Cited: 21
Myocardial Fibrosis in Hypertrophic Cardiomyopathy: A Perspective from Fibroblasts
Maja Schlittler, Peter P. Pramstaller, Alessandra Rossini, et al.
International Journal of Molecular Sciences (2023) Vol. 24, Iss. 19, pp. 14845-14845
Open Access | Times Cited: 21
Mechanism of YY1 mediating autophagy dependent ferroptosis in PM2.5 induced cardiac fibrosis
Huaifang Hu, Lipeng Li, Huaxing Zhang, et al.
Chemosphere (2023) Vol. 315, pp. 137749-137749
Closed Access | Times Cited: 20
Exercise-induced circular RNA circUtrn is required for cardiac physiological hypertrophy and prevents myocardial ischaemia–reperfusion injury
Lijun Wang, Jingyi Feng, Feng Xing, et al.
Cardiovascular Research (2023) Vol. 119, Iss. 16, pp. 2638-2652
Closed Access | Times Cited: 20

Scroll to top