OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

The licorice flavonoid isoliquiritigenin attenuates Mycobacterium tuberculosis-induced inflammation through Notch1/NF-κB and MAPK signaling pathways
Jinxia Sun, Qingwen Zhang, Gui‐Zhen Yang, et al.
Journal of Ethnopharmacology (2022) Vol. 294, pp. 115368-115368
Closed Access | Times Cited: 33

Showing 26-50 of 33 citing articles:

Effect of SiHuangQingXinWan on Klebsiella pneumoniae-induced pneumonia: mechanistic insights
He Liu, Xiaoluo Sun, Sian Tao, et al.
Frontiers in Pharmacology (2024) Vol. 15
Open Access

Biological insights and therapeutic potential of Glycyrrhiza uralensis and its bioactive compounds: an updated review
Sibhghatulla Shaikh, Khurshid Ahmad, Jeong Ho Lim, et al.
Archives of Pharmacal Research (2024)
Closed Access

Unveiling the Essence of Isoliquiritigenin: Exploring Its Chemistry, Pharmacokinetics, and Pharmacological Potential
P. Mohamed Nihal, Debasish Mohapatra, Alam Mohd Adil Alam Manir, et al.
Chemistry Africa (2024)
Closed Access

Qingfei Paidu Decoction Inhibits LPS-induced Acute Lung Injury by Targeting the Complement Pathway
Dandan Shi, Luna Ge, Lei Yan, et al.
Exploratory Research and Hypothesis in Medicine (2023) Vol. 000, Iss. 000, pp. 000-000
Open Access | Times Cited: 1

Research Progress on Pathogenesis of Spinal Tuberculosis and Anti-Tuberculosis Mechanism of Traditional Chinese Medicine
鹤阳 黄
Traditional Chinese Medicine (2023) Vol. 12, Iss. 10, pp. 3137-3145
Closed Access

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