OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

The JAK-STAT Pathway: Impact on Human Disease and Therapeutic Intervention
John J. O’Shea, Daniella M. Schwartz, Alejandro V. Villarino, et al.
Annual Review of Medicine (2015) Vol. 66, Iss. 1, pp. 311-328
Open Access | Times Cited: 1322

Showing 1-25 of 1322 citing articles:

Inflammatory responses and inflammation-associated diseases in organs
Linlin Chen, Huidan Deng, Hengmin Cui, et al.
Oncotarget (2017) Vol. 9, Iss. 6, pp. 7204-7218
Open Access | Times Cited: 3966

Rheumatoid arthritis
Josef S Smolen, Daniel Aletaha, Iain B. McInnes
The Lancet (2016) Vol. 388, Iss. 10055, pp. 2023-2038
Open Access | Times Cited: 3691

The JAK/STAT signaling pathway: from bench to clinic
Xiaoyi Hu, Jing Li, Maorong Fu, et al.
Signal Transduction and Targeted Therapy (2021) Vol. 6, Iss. 1
Open Access | Times Cited: 1422

Small molecules in targeted cancer therapy: advances, challenges, and future perspectives
Lei Zhong, Yueshan Li, Liang Xiong, et al.
Signal Transduction and Targeted Therapy (2021) Vol. 6, Iss. 1
Open Access | Times Cited: 1127

JAK inhibition as a therapeutic strategy for immune and inflammatory diseases
Daniella M. Schwartz, Yuka Kanno, Alejandro V. Villarino, et al.
Nature Reviews Drug Discovery (2017) Vol. 16, Iss. 12, pp. 843-862
Open Access | Times Cited: 1021

Mechanisms and consequences of Jak–STAT signaling in the immune system
Alejandro V. Villarino, Yuka Kanno, John J. O′Shea
Nature Immunology (2017) Vol. 18, Iss. 4, pp. 374-384
Open Access | Times Cited: 1005

JAK–STAT Signaling as a Target for Inflammatory and Autoimmune Diseases: Current and Future Prospects
Shubhasree Banerjee, Ann Biehl, Massimo Gadina, et al.
Drugs (2017) Vol. 77, Iss. 5, pp. 521-546
Open Access | Times Cited: 937

Mechanisms of Disease: Inflammatory Bowel Diseases
Guilherme Piovezani Ramos, Konstantinos A. Papadakis
Mayo Clinic Proceedings (2019) Vol. 94, Iss. 1, pp. 155-165
Open Access | Times Cited: 772

Signaling and Function of Interleukin-2 in T Lymphocytes
Sarah H. Ross, Doreen A. Cantrell
Annual Review of Immunology (2018) Vol. 36, Iss. 1, pp. 411-433
Open Access | Times Cited: 743

The role of JAK/STAT signaling pathway and its inhibitors in diseases
Ping Xin, Xiaoyun Xu, Chengjie Deng, et al.
International Immunopharmacology (2020) Vol. 80, pp. 106210-106210
Closed Access | Times Cited: 705

The role of JAK-STAT signaling pathway and its regulators in the fate of T helper cells
Farhad Seif, Majid Khoshmirsafa, Hossein Aazami, et al.
Cell Communication and Signaling (2017) Vol. 15, Iss. 1
Open Access | Times Cited: 686

Limiting inflammation—the negative regulation of NF-κB and the NLRP3 inflammasome
Inna S. Afonina, Zhenyu Zhong, Michael Karin, et al.
Nature Immunology (2017) Vol. 18, Iss. 8, pp. 861-869
Closed Access | Times Cited: 653

Erratum to: JAK–STAT Signaling as a Target for Inflammatory and Autoimmune Diseases: Current and Future Prospects
Shubhasree Banerjee, Ann Biehl, Massimo Gadina, et al.
Drugs (2017) Vol. 77, Iss. 8, pp. 939-939
Open Access | Times Cited: 574

Type I/II cytokines, JAKs, and new strategies for treating autoimmune diseases
Daniella M. Schwartz, Michael Bonelli, Massimo Gadina, et al.
Nature Reviews Rheumatology (2015) Vol. 12, Iss. 1, pp. 25-36
Open Access | Times Cited: 552

Neutrophils as emerging therapeutic targets
Tamás Németh, Markus Sperandio, Attila Mócsai
Nature Reviews Drug Discovery (2020) Vol. 19, Iss. 4, pp. 253-275
Closed Access | Times Cited: 540

Epidermal growth factor receptor and EGFRvIII in glioblastoma: signaling pathways and targeted therapies
Zhenyi An, Ozlem Aksoy, Tina Zheng, et al.
Oncogene (2018) Vol. 37, Iss. 12, pp. 1561-1575
Open Access | Times Cited: 507

Heavy metal associated health hazards: An interplay of oxidative stress and signal transduction
Jagdish Gopal Paithankar, Sanjay Saini, Shiwangi Dwivedi, et al.
Chemosphere (2020) Vol. 262, pp. 128350-128350
Closed Access | Times Cited: 459

Harnessing the plasticity of CD4+ T cells to treat immune-mediated disease
Michel DuPage, Jeffrey A. Bluestone
Nature reviews. Immunology (2016) Vol. 16, Iss. 3, pp. 149-163
Closed Access | Times Cited: 444

Therapeutically exploiting STAT3 activity in cancer — using tissue repair as a road map
Jennifer Huynh, Ashwini L. Chand, Daniel J. Gough, et al.
Nature reviews. Cancer (2018) Vol. 19, Iss. 2, pp. 82-96
Closed Access | Times Cited: 444

JAK inhibitors in dermatology: The promise of a new drug class
William Damsky, Brett King
Journal of the American Academy of Dermatology (2017) Vol. 76, Iss. 4, pp. 736-744
Open Access | Times Cited: 427

Upadacitinib Versus Placebo or Adalimumab in Patients With Rheumatoid Arthritis and an Inadequate Response to Methotrexate: Results of a Phase III, Double‐Blind, Randomized Controlled Trial
Roy Fleischmann, Aileen L. Pangan, In‐Ho Song, et al.
Arthritis & Rheumatology (2019) Vol. 71, Iss. 11, pp. 1788-1800
Closed Access | Times Cited: 356

Erratum: JAK inhibition as a therapeutic strategy for immune and inflammatory diseases
Daniella M. Schwartz, Yuka Kanno, Alejandro V. Villarino, et al.
Nature Reviews Drug Discovery (2017) Vol. 17, Iss. 1, pp. 78-78
Open Access | Times Cited: 315

The potential and controversy of targeting STAT family members in cancer
Yannick Verhoeven, Sam Tilborghs, Julie Jacobs, et al.
Seminars in Cancer Biology (2019) Vol. 60, pp. 41-56
Open Access | Times Cited: 311

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